Duct arteriosus [Ductus Arteriosus, Pna; Ductus Arteriosus (Botalli), Bna; Syn. Botallov Proto]

The ductus arteriosus (Ductus arteriosus) is a blood vessel that connects the pulmonary trunk and the aorta in the fetus. It is formed from the left sixth branchial arch and after birth quickly disappears into a fibrous cord. However, if this duct does not close completely, it can lead to the development of congenital heart disease - patent ductus arteriosus.

The ductus arteriosus plays an important role in the development of the fetal cardiovascular system. It ensures blood flow to the lungs and heart, and also supplies oxygen and nutrients to the tissues and organs of the fetus. In addition, the ductus arteriosus helps regulate blood pressure in the fetus, which is important for its normal development.

However, after birth, when the placenta and umbilical cord cease to perform their functions, the ductus arteriosus begins to quickly close and reduce to a fibrous cord. If this process does not occur completely, a congenital heart defect called patent ductus arteriosus (PDA) may develop.

PDA is a condition in which the ductus arteriosus remains open and continues to function after birth. This can lead to various complications such as arterial hypertension, pulmonary hypertension, heart failure and others.

Treatment for PDA depends on the severity of the disease and may include surgery, drug therapy, or a combination of these methods. It is important to note that PDA is a serious disease that requires timely treatment.

According to ICD 10, the arterial proton has code Q23. The origin of the anomaly is to blame for the gene with the code POS, which is responsible for lipid metabolism. The interpretation of the abbreviations is as follows: Q23 “Ductus arteriosus” (DCA) is an underdeveloped vascular canal located in the mediastinum, which arose instead of the branchial arterial duct, which connects the aorta and the pulmonary artery of the fetus. May cause pathologies in the development of the cardiovascular system. The disease happens:

open. Accompanied by the presence of a compaction in the center of the chest at the site of communication between the aortas. Residues last a lifetime;

closed. The canal is overgrown, there are no signs of disease. Heart defects only remain in the anamnesis.