Hepatonephrosis and its consequences **Hepatonephrosis** is a violation of the anatomy and functionality of the kidneys and liver, which can lead to serious consequences. It occurs due to mixing of blood in the gastrointestinal tract or abdominal cavity.
**Symptoms of hepatonephrosis** are nausea, vomiting, weakness, abdominal pain, pale skin and yellowness of the skin and mucous membranes. In severe cases, in the absence of timely medical attention, seizures, coma and death may develop.
The main **causes of hepatonephrosis** include hypoxia due to poisoning by respiratory products entering the blood through the stomach, diaphragm or esophagus, injuries with hematoma, tumors and infectious diseases. The hematoma can be either long-standing or recently occurring, after which hepatonephrasia often occurs.
Risk factors include anaembryogenesis and other congenital anomas
Hepatonephrosis is a pathological condition characterized by a combination of splenomegaly, characteristic jaundice and renal failure, associated with impaired patency of the lymphatic vessels of the liver, including small portosystemic vessels with secondary involvement of the bile ducts and impaired outflow of bile with subsequent biliary dysfunction and biochemical disorders. Hepatonephrosis is a complication of many diseases (heart, lungs, digestive, endocrine systems, etc.).