Neuropathy Reticular Sensory Hereditary

**Hereditary sensory reticular neuropathy** (hereditary sensory retinoneuropathy), also known as Hyxa disease, is a rare inherited disease that causes loss of sensation in multiple senses, including vision, hearing, smell, taste and touch. This problem usually occurs in young people between the ages of 20 and 40, but can occur later in life. Doctors may call this disorder because of its connection to heredity and the nervous system. This study will examine the causes, symptoms, treatment and prognosis of hereditary reticular sensory neuropathy.

Symptoms of hereditary sensitive retinoneuropathy Most often, hereditary sensitive retinal neuropathy manifests itself in the form of vision loss. This may be caused by swelling of the optic nerve, which results in neglect of the optic nerve pulposus. Symptoms may also include blind spots, spots in the central field of vision, limited visibility, blurred vision, difficulty reading small print and other text, and difficulty recognizing shapes, colors, and shades of objects and surfaces in the environment.

Treatment Therapy for hereditary sensory retinoneuritis is carried out by restoring damaged nerves and increasing blood supply, rehabilitation, drug support and ongoing counseling. The most effective treatments for hereditary retinoneuritis disorders include surgical interventions, certain orthopedic assistive devices, and surgical procedures with a low risk of injury. Bone auditory music and optical occlusion are among a number of music therapy and auditory acoustics techniques that have been effective, although the relationship between them and neuropathy is unclear. Retinoneuritis can also often lead to the intervention of medical devices such as hearing aids and orthopedic devices such as special large-field glasses.

Prognosis and prevention of hereditary sensory retinoneuritis Prognosis for hereditary sensoretino-vaginal disorders in children remains disappointing. Most children with this disorder lose their vision permanently and are subsequently unable to participate in social activities. The prognosis may be further worsened if any part of the eye responsible for vision is covered with scar tissue after the initial disease. Prevention of hereditary pathological accumulation of scars helps preserve vision. Research is ongoing to determine which procedures increase the chance of preserving vision or restoring retinal and optic nerve function in adults with neuropathic hereditary retinoneuria. Although early symptoms are easily detectable and reversible, most cases of inherited Retinoxanl occur during adolescence or the first years of life. Many children recover their vision within the first few years after diagnosis, but for others it is permanent. Frequent occurrence of jaundice, high blood pressure, diabetes or high cholesterol are factors associated with hereditary retinoneloria and therefore need special attention.