Recklinghausen Dysontogenetic Dystrophy

Recklinghausen's Dysostogenic Dystrophy Dysostogenes Recklinghausen, or "dysostogenic dystoric Recklinghaus syndrome" (abbreviated as DRD) is a rare hereditary disease characterized by impaired development of bones and bones, as well as other abnormalities. There are several synonyms for this disease: “Recklingaus syndrome”, “Dysostogenesis type A”, “Recklingaus and achondroplasia syndromes”, etc. In this article we will look at the description of this disease, its causes, symptoms and treatment methods.

General information about the disease First, let's define what dysostogenerans are. Dysostogenic dystogenics are a rare metabolic disorder manifested by abnormalities of bone development or osteogenesis. The disease occurs due to defects in certain genes and proteins that are involved in the proper formation of bone tissue. This leads to change

Recklinghauen Dyzontogenetic syndrome Recklinghauen Dyzontogenic Dystrogia Recklinghausen developed an atypical type of ozone triplex longitudinal monopolar dysostosis with a hernia with an oval and angular arch defect - Recklinghauen dysontogeny. These are diseases of the spine and impaired development of the skull and limbs.

Anatomy of the disease Secondary defect of the bony conus associated with the replacing hyperosteum, starting primarily in the thoracic region, leading to wear and tear of the motor muscles of the back and respiratory system, causing respiratory disorders during perfusion of the spinal cord, i.e. weakness of the lower extremities, imbalance, forming curvature of the spine .

The penetration of cerebrospinal fluid through the clefts of the subcutaneous muscular layer of the pelvic floor leads to tumors of the buttocks, inguinal cavities and genitals in children. A defect in the septum between the transverse obturator, sigmoid bone cord, pancreas, liver and left kidney diagnoses somatogenic tumor diseases. Boys who are first born suffer earlier than average, boys who are in average condition operate and restore average defects within 24 hours. In girls, the recovery period is extended by several months. The prognosis is possible after surgical treatment.

Assessing a child’s postural abnormalities allows us to determine the priority reason for testing if deformities of the spine and legs occur before the age of 5. Typically, this period is unfavorable for surgical treatment of secondary forms of cerebral palsy, since motor functions have developed, and the skeletal balance has not yet been fully determined. For the treatment of children with secondary