Atrophy of the pupillary nerve is damage to the optic (optic) nerve, characterized by its degeneration, hypoplasia, and loss of conduction along its fibers. Another name for ADN is “optic neuritis.”
The main cause of partial atrophy of the optic nerve is alcoholic and diabetic neuropathy. Common causes of WGA are a number of common chronic diseases, inflammatory and infectious processes, circulatory disorders in the area of the optic nerves, as well as the use of toxic agents (for example, medications). ADPN occurs when the overall tone of the body decreases and there is a deficiency of vitamins B1 and B6. Among the rare causes of ADN are trauma to the eyeball, tumors of the brain, vascular insufficiency, and oncological diseases of the ophthalmological and otolaryngological profile. The examination of the optic nerve and retina of the eye is carried out by an ophthalmologist; special preparation is not required.
1. Drug therapy for alcoholic optic neuritis When alcohol enters the human body, it first passes through the liver, where it is metabolized into various metabolites. Some of them have a depressing effect on the nervous system as a whole. Alcohol, having passed the ethanol stage, remains in the blood in free or bound form due to albumin. A certain amount of acetaldehyde is formed along the metabolic chain “ethyl alcohol→acetaldehyde→acetic acid.” The latter, after being excreted from the human body, ends up in uric acid, that is, all this metabolic