Hypoglycemia Spontaneous Idiopathic

Hypoglycemic spontaneous idiopathic syndrome is a complex of symptoms associated with a short-term decrease in blood glucose levels, the cause of which is unclear or unknown. Such a breakdown of sensitivity in the brain may be due to other reasons (for example, stroke, the development of thrombosis). In most cases, this disease is discovered by chance. The occurrence of hypoglycemia provokes increased production of adrenaline and cortisol.

Pathogenesis of the disease

A decrease in glucose concentration occurs due to increased production of adrenaline, as well as inhibition of glycogen synthesis. Adrenaline initially has a hypoglycemic effect, and subsequently glucose is formed in the liver.

Hypoglycemia is a decrease in blood glucose levels to critical values ​​(usually less than 2.2-3.3 mmol/l or 40-60 mg/dl). It is characterized by the appearance of tremors, sweating, hunger, weakness, headache, pale skin, rapid heartbeat, and decreased blood pressure.

Under the influence of provoking factors, the blood sugar level decreases to a value below the threshold of 3.3 mmol/l. After this, an attack of hypoglycemia occurs. Provoking factors, in most cases, are low calorie intake of food (carbohydrates), alcohol intoxication, and side effects of medications. Very often, the hypoglycemic form is observed in patients who eat food or do not pay the necessary attention to their health, or who violate the rules of use and dosage of the diet recommended for pathology of the kidneys and endocrine glands.

Complications of type 2 diabetes mellitus

Hypoglycemic syndrome can be a consequence of a disease, but in some cases it occurs independently. The specific term for this condition is “spontaneous hypoglycemia.” Hypoglycemic syndrome, or diabetic