Spondylopathy tabetica

Tabetic spondylopathy: causes, symptoms and treatment

Tabetic spondylopathy is a rare disease that is caused by damage to the spinal cord and nerve roots responsible for movement and sensation in the limbs. This disease can occur as a complication of syphilis, but most often it is associated with tabes, which is one of the manifestations of neurosyphilis.

With Tabetic spondylopathy, damage to the spinal cord and roots occurs, which leads to various disorders of motor and sensory functions. Symptoms include severe pain in the back and limbs, loss of coordination, seizures, problems with urination and bowel movements. In addition, patients may experience vision, hearing, and speech impairments.

To diagnose Tabetic spondylopathy, various research methods are used, including magnetic resonance imaging (MRI) and computed tomography (CT). Based on the results of the study, the doctor can make a diagnosis and prescribe treatment.

Treatment of tabetic spondylopathy is aimed at eliminating symptoms and slowing the progression of the disease. For this purpose, anticonvulsants, antidepressants, drugs to strengthen bones and many other drugs may be prescribed. In addition, it is important to provide the patient with proper care and rehabilitation, including physical therapy and special exercises.

Despite the fact that tabetic spondylopathy is a serious disease, modern medicine can significantly improve the quality of life of patients and slow down the progression of the disease. It is important to consult a doctor promptly when the first symptoms appear and follow his instructions in order to achieve the maximum effect of treatment.



The spondylopathic or spondylogenic lumbosacral form of diabetic neuropathy was named after the “tabetic” symptoms and syndromes of sensory disturbances, reflexes and sometimes pelvic organ function. The name "spondylopathy" is borrowed from the Greek - "lumbar". Officially presented in 2009, the clinical concept suggests that the spondylopathic form of neuropathy is preceded, occurs in parallel, or sometimes develops manifestly in the early stages of the underlying disease - neural anosmtia (decreased sensitivity) of the nerve trunks.