Trichoptilosis

Trichoptilosis: causes, symptoms and treatment

Trichoptilosis, also known as trichoschisis, is a hair condition in which the ends of the hair split and break. This leads to hair looking uneven and unkempt. In this article we will look at the causes, symptoms and treatment of trichoptilosis.

Causes of trichoptilosis

There are several reasons why hair may begin to split and break. One of the most common factors is improper hair care. This may include frequent use of hot hair styling tools such as blow dryers, curling irons and flat irons. Also, improper use of brushes and combs can lead to trichoptilosis.

Another factor that can lead to trichoptilosis is a lack of nutrients in the body. Hair needs certain vitamins and minerals such as vitamin A, vitamin E, iron and zinc to stay healthy and strong. A lack of these substances can lead to trichoptilosis.

Symptoms of trichoptilosis

The main symptom of trichoptilosis is the appearance of split and brittle hair ends. Hair may also appear dry and brittle throughout its entire length. In some cases, the hair may have an unnatural shine and look awkward when styled.

Treatment of trichoptilosis

Treatment for trichoptilosis may include changing the way you care for your hair. This may mean cutting back on the use of hot hair styling tools or using brushes and combs with soft bristles. It is also important to eat right to provide your hair with the nutrients it needs.

In some cases, it may be necessary to trim off cut or brittle hair ends. This will help prevent further hair splitting and promote hair health and beauty.

In conclusion, trichoptilosis is a common hair condition that can be caused by improper hair care or lack of nutrients in the body. If you notice that your hair has started to split and break, it is recommended that you contact a hair care specialist or trichologist for advice on treating trichoptilosis.



Trichoptilosis is one of the acantholytic dermatoses of unknown etiology. Indurative trichoptylosing glossitis and accompanying autoimmune thyroiditis are often combined with damage to the nervous system (Sjögren's syndrome). Erythematous lesions with a grayish-white tint appear on the mucous membrane of the cheeks, palate and posterior wall of the pharynx; when the discharge is removed, longitudinal folds resembling hair are formed. Subsequently, the lesions ulcerate, cysts or pigment spots appear, sometimes erosions and cracks. Less commonly, regional lymphadenopathy is possible, usually asymmetric. Treatment includes the use of corticosteroids, cytostatics, and retinoids.