Anemia Sideroblastic Secondary

Secondary sideroblastic anemia: causes, symptoms and treatment

Anemia secondary to sideroblastic (ASV) is a rare blood disorder characterized by low red blood cell counts, low hemoglobin levels, and the presence of dysfunctional sideroblast cells in the bone marrow.

Similar to primary sideroblastic anemia, ASV is also associated with impaired hemoglobin synthesis and a decrease in the number of red blood cells in the blood. However, unlike the primary form, ASV develops due to other diseases or factors, including alcoholism, chronic infections, tumors, autoimmune diseases and long-term use of certain medications.

Symptoms of ASV may include weakness, fatigue, pale skin and mucous membranes, short-term blurred vision, red tongue, respiratory problems, and increased sensitivity to cold. Patients may also experience an enlarged liver and spleen.

Diagnosis of ASV requires blood testing and bone marrow biopsy. Blood tests reveal low hemoglobin levels, elevated iron levels, and the presence of sideroblast cells in the bone marrow that are unable to function normally. A bone marrow biopsy can identify the cause of ASV.

Treatment for ASV depends on the causes that led to its development. In some cases, a blood transfusion may be needed to increase the level of red blood cells in the blood. Drugs, surgery, or other treatments may be prescribed to treat the underlying condition that caused ASV.

Overall, ASV is a serious disease that requires timely diagnosis and treatment. If you suspect the development of this disease, you must consult a doctor to conduct appropriate examinations and prescribe the necessary treatment.



Secondary sideroblastic anemia is a rare and serious disease caused by disorders in the human hematopoietic system. The concentration of hemoglobin in the blood drops as well as the number of red blood cells, which leads to chronic anemia (decreased hemoglobin amount). When hemoglobin decreases, oxygen starvation of organs and tissues occurs, severe fatigue, weakness, pale skin, deformation of nails and hair structure, frequent thirst and headaches develop. In addition, the formation of hemoglobin does not occur completely and it does not differ from the hemoglobin accumulated in tissues.