Anemia Diphyllobothriasis

Diphyllobothriasis anemia: symptoms, causes and treatment

Diphyllobothrium anemia, also known as botryocephalic anemia or helminthic B12 deficiency anemia, is a rare but serious disease caused by infection with the tapeworm Diphyllobothrium latum. This parasite can lead to impaired absorption of vitamin B12 in the gastrointestinal tract, which leads to the development of anemia.

Symptoms of diphyllobothriasis anemia include general weakness, fatigue, dizziness, shortness of breath, palpitations, pale skin and mucous membranes, as well as decreased appetite and weight loss. In more severe cases, there may be jaundice and damage to the nervous system, leading to numbness and loss of sensation in the limbs.

The cause of diphyllobothrium anemia is associated with the presence of the tapeworm Diphyllobothrium latum in the human intestine. The worm feeds on vitamin B12, and its presence can lead to decreased levels of this vitamin in the human body. This can happen when eating raw or undercooked fish, which is the main source of infection.

Treatment for diphyllobothriasis anemia involves killing the tapeworm and restoring vitamin B12 levels. For this purpose, anthelmintic drugs are used that destroy parasites, as well as drugs containing vitamin B12. With proper treatment, diphyllobothriasis anemia can be completely cured.

Overall, anemia diphyllobothriasis is a rare disease, but can lead to serious consequences for a person's health. Therefore, to prevent infection with tapeworms, it is necessary to eat only well-cooked fish and monitor food hygiene. If you experience symptoms associated with anemia diphyllobothriasis, see your doctor for diagnosis and treatment.

**DIPHYLLOBOTHRIOUS ANEMIA.**

Diphyllobrotriase anemia is an acute or chronic anemia that develops as a result of deficiency of vitamin B12 and its coenzyme (vitamin-like factor of the extrinsic pathway). Sources of B12 are only animal products (meat and offal, fish, eggs).

The acute form of the disease develops with a lack of vitamin B 12 intake from food. The disease can occur 3-4 weeks after surgery on the stomach, duodenum, or resection of the jejunum. Early diagnosis is due to a severe course and symptoms similar to manifestations of dystrophic intestinal processes - damage to the transverse colon and ileum. Specific