Apraxia Akinetic

Akinetic apraxia: Understanding and characteristics

Akinetic apraxia, also known as psychomotor apraxia, is a neurological disorder characterized by an impairment in the planning and execution of intentional movements. This condition is caused by damage to certain areas of the brain, mainly the basal ganglia.

Characteristics of apraxia akinetic include a general loss of motor ability, decreased speed and smoothness of movements, and difficulty initiating and organizing motor programs. Patients with apraxia akineticus may have difficulty performing simple tasks such as getting dressed, washing dishes, or writing. They may exhibit limited range of motion and monotonous behavior.

The cause of akinetic apraxia is often damage to the basal ganglia, which play an important role in the regulation of motor functions. This can be caused by a variety of factors, including illnesses such as Parkinson's disease, strokes, head injuries, or certain medications.

Diagnosing akinetic apraxia can be difficult because its symptoms may overlap with other movement disorders. However, doctors may use a variety of methods, including neurological tests, assessment of motor skills, and examination of the brain through various educational methods.

Treatment for apraxia akineticus is usually aimed at managing the underlying condition that is causing the symptoms. In some cases, medications may be prescribed, such as medications to improve motor function or reduce symptoms of parkinsonism. Physical therapy and rehabilitation may also be helpful in improving motor skills and improving patients' quality of life.

Akinetic apraxia is a complex neurological disorder that affects a person's ability to perform everyday motor tasks. Modern medicine strives to better understand this condition and develop effective treatments to improve the quality of life of patients suffering from apraxia akineticis.



Apraxia is a violation of voluntary purposeful movements and actions, in the absence of paralysis or paresis. When performing a task, difficulties concern both the general program of actions and the details of execution; the emotional-volitional regulation of behavior also suffers. Unlike apraxia of ideation aphasia (aphasic agnosia), apraxic aphasia can be eliminated, which allows for differential diagnostic research. It is usually regarded as ungrammatical. Taking into account the differences in the etiology and localization of speech disorders, motor, efferent and autonomic forms are distinguished. There are known cases of isolated apraxia of the articulatory apparatus. In addition, constative and violent forms are distinguished. Of particular interest is a disorder of the facial muscles, manifested by the absence of facial movements, which is combined with the absence of pronunciation of sounds (motor form). Damage to the frontal lobes leads to “atomization” of the thought process until the onset of complete amentia (efferent, or acoustic)