CORTICAL APRAXIA – a type of apraxia, expressed in a violation of voluntary movements while maintaining speech and intellectual activity
This pathology is quite common and accounts for approximately 5% of all known motor dysfunctions. It is worth noting that it does not have a direct connection with age. In childhood, the percentage is always lower than in adults. Here it is worth noting the greater nervous load that children are exposed to than adults. In addition, childhood is a period of increased activity, which cannot always be implemented correctly.
Apraxia of the cerebral cortex.
By definition, apraxia is a disorder of voluntary types of movements, combined with the preservation of speech communication and consciousness (Diseases of the nervous system. Edited by Academician N.N. Yakhno - 3rd ed., - M.: Medicine, 2005). With this pathology, various disturbances of spatial orientation, locomotor acts, actions with objects, writing, dressing both as a whole and parts of the body, voluntary urination and defecation can be observed. Possible disorder of voluntary change of postures. Accordingly, the manifestations of apraxia can be motor and non-motor. The actual derivative of the segmental apparatus of the central nervous system, which includes damage and deformation of the brain for various reasons, are the reasons for the localization of disorders, which depend on their morphological changes. Thus, loss of reflexes and sensitivity, decrease or loss of muscle tone, an increase in the paresis reaction, paroxysmal movements, dysmetria, distortion of existing reflexes, loss of the ability to balance, and impaired muscle coordination may prevail. Leading role
Apraxia, affectaphoria are symptoms of a violation of higher brain functions in the form of deautomatization of complex movements and loss of their intentional nature while maintaining motor programs. Examples of syndromes with an apraxia component are cortical deautomatization of movements, psychomotor areflexia.
The description of this disease is described in group 6 of symptoms of acute poisoning by hypnotics. It occurs as a result of disconnection of the cerebral cortex and subcortical formations due to neuroinfection and metabolic disorders. It is considered an iatrogenic symptom for most patients in the acute period of poisoning with psychotropic drugs. Often found in stroke, craniocerebral trauma