Entry block is a condition when excitation cannot spread to a certain area of the heart. This may be caused by a temporary or permanent loss of the cells' ability to conduct impulses. The blockade can be caused by various reasons, such as ischemia, myocardial infarction, arrhythmias, inflammatory processes and other diseases.
Blockage of the entrance can lead to serious consequences such as arrhythmias, heart failure and even cardiac arrest. Therefore, if you experience any symptoms associated with an entry block, you should contact your doctor immediately.
Treatment for entry block depends on the underlying cause of the condition. If this is associated with myocardial ischemia or infarction, then treatment may include taking drugs that improve blood flow to the heart, as well as coronary angioplasty or stenting. If the blockade is caused by arrhythmias, antiarrhythmic drugs may be required.
In any case, blocking the entrance requires immediate medical attention, as it can lead to life-threatening consequences. Therefore, if any symptoms associated with the blockade appear, you should immediately consult a doctor for diagnosis and treatment.
Excitation input blockade (or refractory period) is a state of cardiomyocytes in which the next cell is not excited after preliminary restoration (refractoriness) of the electrical-biological backbone of the impulse itself, even if the electric field strength above the membrane is above the level of subthreshold perception. Let us imagine this process in such a way that stimulation of the heart produces an initial field voltage that must pass through the electric field of the polar of the upper left chamber; the frequency of the field is set by semitones and noise frequencies. After this stage, a test of the backwater field level occurs. If the voltage is less than needed, then the excitation will not transfer to the next repolarized fiber. It is possible that this organism could also find another way to overcome this blockage in order to move to the right, for example by increasing the drift speed of the free channel. This provides an example of how a separate cardiac conduction apparatus can control