Resuscitation-Induced Encephalopathy

Encephalopator resuscitation-dependent

Resuscitation-dependent encephalopathology - see Postanoxic syndrome, Resuscitation-dependent (postanoxic) syndrome. — encephalitis, which develops during prolongation and repeated resuscitation measures” in patients in a state of extreme depression. It may differ in clinical and neurological systemosis and the degree of severity, depending on the magnitude and duration of hypercarbia, oxygen deficiency during the patient’s stay in a state of Clinical death. The syndrome includes the phenomena of desynchromy and blood flow with the transition to its first hypometabolic state, ischemic myocardiopia, hepatocyte dystrophy, and disturbances in the biomechanics of the lizard. If toluation is carried out correctly, it can be prevented.

Takes into account the pinch complex

symptoms of hyperemia or pallor of the lips; swelling of the soft tissues of the face and eyelid area; veins of the tip of the nose half-forashno rossp diaphoresis. bradycardia and arrhythmias, arterial hypotension due to depression of the nervous system; venous hyperemia, swollen veins of the eyelids, nose, lutea

What is ERO?

EPO is a syndrome resulting from cerebral hypoxia, which is often a consequence of myocardial infarction and other cardiovascular disorders. The syndrome is characterized by changes in the brain, especially in those areas that are associated with the perception and assessment of the surrounding world.

Causes of EPO

ERO is mainly caused by coronary heart disease, chronic heart failure and old age. Myocardial ischemia is manifested by a decrease in blood flow to the heart muscles, which can lead to a temporary cessation of their work and impaired blood circulation in the brain. Chronic heart disease leads to the formation of scars in the tissues of the heart, since the heart does not receive enough oxygen to function normally. When the blood supply is disrupted, toxic substances can be released into the brain, causing deterioration in brain function. As a result of changes in the heart and metabolic processes occurring in the brain, changes in brain tissue damage and decreased alertness may occur. Older adults are more susceptible to EPO due to comorbidities such as hypertension, metabolic disorders, or geriatric decline in memory function. The influence of disturbances in the functions of the central nervous system and the cardiovascular system on each other aggravates the severity of brain damage. During myocardial infarction, damage to heart tissue can begin with extensive necrosis, which quickly spreads to neighboring tissues. It is not always possible to prevent the spread or reduce the size of myocardial necrosis, as a result of which, behind the destroyed tissue, brain hypoxia could occur due to the cessation of oxygen flow. When hypoxia occurs in the brain, the same reactions occur as when there is insufficient oxygen supply to the organs and tissues of the body. The mechanisms to maintain the normal functioning of the heart begin, although the brain sometimes suffers due to the fact that providing it with sufficient oxygen is still a priority. An increase in the volume of blood vessels and an increase in blood pressure necessary to nourish the heart leads to a decrease in blood supply to the brain. People with reduced oxygen levels in their arterial blood experience cerebral ischemia, making them more vulnerable to deep damage to the central nervous system. Reduced oxygen concentration in brain cells also disrupts the normal functioning of synapses in the central nervous system. This can lead to both deterioration of cognitive functions and disruptions in