Haglund-Schinz Disease

Haglund-Schinz Disease: symptoms, causes and treatment

Haglund-Schinz disease, also known as osteochondrosis of the calcaneal apophysis, is a common condition that often occurs in athletes and people who wear tight shoes. This condition causes inflammation of the heel bone and surrounding tissue, resulting in rearfoot and heel pain.

Haglund-Schinz disease was named after two surgeons: Swedish orthopedist Per-Isak Haglund and German surgeon Robert Schintz. They described this condition in the late 19th and early 20th centuries.

The causes of Haglund-Schinz disease are not entirely clear, but, as a rule, its development is associated with repeated trauma to the heel, for example, during active sports or wearing uncomfortable shoes with a hard back. Also, the risk of the disease increases with flat feet, abnormal development of the foot or deformation of the legs.

Symptoms of Haglund-Schinz disease include pain in the back of the foot and heel, which may worsen when walking or running or when putting pressure on the heel. Patients may also experience swelling and redness near the heel, as well as burning or numbness in the back of the foot.

Diagnosis of the disease may require x-rays, ultrasound, or magnetic resonance imaging. Treatment for Haglund-Schinz disease includes rest and limited physical activity, ice application, and continued wearing of soft-backed shoes. In some cases, medications may be required to relieve pain and inflammation, as well as physical therapy or surgery.

Haglund-Schinz disease can be an unpleasant and painful condition, but with proper treatment and care, most patients overcome the disease and return to their normal lives and activities. If you experience symptoms of Haglund-Schinz disease, consult your doctor for diagnosis and treatment.

Haglund - Schintz's disease (arthrosis of the vertebrae, osteoarthritis of the finger, dissection of Forestami syndrome, Hog syndrome, plantar arthritis) is a disease of the musculoskeletal system, characterized by impaired functioning of the vertebral brake. The development of the disease is associated with age-related changes in osteoclasts, matrix damage, cartilage and bone displacement and protrusion, muscle spasm and ligament deformation. There is a contracture of the lower leg and the palatine part of the foot with physical and painful blockade of the lower extremities. Pathomorphological changes of the disease are characterized by a concave-recessive transformation of cartilaginous tissues with the accumulation of pigments on the articular nucleus, formed by a degenerative-dystrophic process, hypertrophy of the fissure apophysis, discirculation of collateral vessels, accumulation of inflammatory contents around neoplasms, and excess mucosa of alveolar parenchyma and thin crust are also included.