Leptomeningitis Adhesive

Leptomeningitis adhesive: Main characteristics and treatment

Leptomeningitis adhesive, also known as leptomeningitis productive or leptomeningitis fibrosing, is a serious inflammatory disease that affects the soft lining of the brain and spinal cord. This condition is characterized by the formation of adhesions (adhesions) between the meningeal membranes, causing gross fibrosis and compaction between them.

The main symptoms of leptomeningitis adhesive include headache, neck stiffness, fever, nausea and vomiting, seizures and impaired consciousness. Symptoms associated with damage to the nervous system, such as weakness, numbness, changes in sensation and loss of coordination, may also occur.

The causes of adhesive leptomeningitis can be varied. One cause is an infection caused by bacteria or viruses, such as meningococcus, herpes simplex virus, or the common cold virus. Other factors such as trauma, tumors or autoimmune diseases are also possible.

The diagnosis of leptomeningitis adhesive is usually based on clinical symptoms, cerebrospinal fluid (CSF), and magnetic resonance imaging (MRI) or computed tomography (CT) scans of the brain and spinal cord. CSF fluid obtained by lumbar puncture may show increased protein levels and changes in cell composition.

Treatment for leptomeningitis adhesive usually involves the use of antibiotics or antiviral drugs to fight infection if it is causing the disease. Corticosteroids may be used to reduce inflammation and reduce symptoms. In some cases, surgery may be required to break the adhesions and restore normal blood flow between the meningeal layers.

The prognosis for patients with leptomeningitis adhesive depends on many factors, including the cause of the disease, timely diagnosis and treatment, and the presence of complications. Timely diagnosis and adequate treatment can significantly improve the prognosis and reduce the risk of complications.

In general, leptomeningitis adhesive is a serious disease that requires medical intervention and specialist supervision. If you suspect leptomeningitis adhesive or are experiencing characteristic symptoms, it is important to see your doctor for an accurate diagnosis and appropriate treatment.

Although leptomeningitis adhesive is a rare condition, general awareness of it can help you recognize symptoms and seek prompt medical attention. Regular consultation with your doctor and following infection prevention guidelines can help reduce your risk of this and other inflammatory diseases of the nervous system.

It is important to note that this article is for informational purposes only and is not a substitute for professional medical advice. If you suspect leptomeningitis adhesive or any other disease, it is recommended that you consult a qualified physician for diagnosis and treatment.



Leptomeningitis is an inflammatory disease of the meninges.

A. A. Bogorodsky identified 2 types of leptomeningitis: unproductive and productive. The cause of the first type is a primary infection from local foci, the second is an infection that has penetrated the skull through the vascular bed (meningitis) or hematogenously (chronic sepsis).

Another type of leptomenigovta is adhesive. There are three forms of adhesive meningitis. All of them have the following in common: the lesion occurs submucosally. , are accompanied by severe endoarachnoid edema, effusion and compression. In the cranial cavity, the periflow of brain substance formed along the meningo-encephaloglya. CSF dynamics are disrupted due to the inflammatory process. To analyze this form, it is worth paying attention to the following with their data: the picture of brain matter changes slightly. An external pineal cyst often occurs. Changes on MRI are not as pronounced as those described for intracranial hematoma. In the acute form, internal hemorrhage begins to occur, intracranial pressure increases, as a result of which there is a pronounced picture of Gummiror. On MRI, the size of the brain decreases in the same way as with meningitis. The development of hydrocephalus is common. With impaired cerebrospinal fluid dynamics, there is an increase in the volume of the lateral ventricles (external cyst up to 6 cm in diameter), sometimes they also begin to die in size (ventriculo-subperiosteal block). If hydrocephalus occurs proactively, then adipose tissue forms between the joint and the layer of fused bone tissue; because of this, the roof of the joint cannot break through since it is closed by this tissue. It is worth noting that to the endplate of the PS