Eye and Tongue Symptom

The eye and tongue symptom is one of the common symptoms that can indicate the presence of various diseases. It manifests itself in the patient's inability to simultaneously keep his eyes closed and his tongue out. This symptom has its own name - “lip-ocular syndrome”.

The eye-tongue symptom occurs in many diseases that affect the facial muscles. For example, minor chorea, choreorrhexis, progressive supranuclear ophthalmoplegia, facial dystonia, etc. Minor chorea is a condition that is caused by dysfunction of the brain. Symptoms of minor choreoresis may include the inability to hold the tongue and eyes simultaneously. This condition may also be accompanied by tremors, seizures and loss of control over muscle movements.

Choreorrhexis is a neurological condition in which the patient experiences incoordination of eye and head movements. This may manifest itself as poor quality of vision, difficulty determining the direction of gaze, and other problems related to eye coordination. Chorera is a fairly rare disease, but it happens that it is combined with other neurological diseases. Chorea may appear on only one side of the body, or affect both sides.

In some cases, symptoms of eye-tongue may include blurred vision, headache, and facial cramps. If you notice such a symptom in your life or in the life of someone close to you, consult a doctor, as it may indicate the presence of some serious diseases.



Eye-tongue symptom “Eye-tongue symptom” is the patient’s inability to keep intact the ocular and other proprioceptive reflexes, the simultaneous onset of which, normally, is noted at the moment when he begins to perform a voluntary motor act. When the cortical parts of the monovisual area are damaged, the patient’s ability to perform certain actions according to instructions on the left or right is lost.

The eye-tongue symptom can occur against the background of headaches and migraines. Patients may also complain of anxiety and restlessness - all this is associated with a violation of the functional state of the Meynert nucleus and striatal homunculus. Deficiency of nerve impulses in the reticular form