Thromboendarterectomy is a surgical operation to remove an atherosclerotic plaque from the lumen of an artery along with a blood clot.
This operation is performed to restore normal blood flow in case of significant narrowing or complete occlusion of the artery due to atherosclerotic plaques and thrombosis. Typically, thromboendarterectomy is performed on the carotid, iliac, femoral and other large arteries.
During the operation, the surgeon makes a longitudinal incision in the artery wall and removes the atherosclerotic plaque along with the blood clot. The artery is then sutured or a patch is sewn in to restore its lumen.
Thromboendarterectomy can effectively restore blood flow and prevent stroke, heart attack and other complications of atherosclerosis. The operation is performed using both open and endovascular approaches.
Overall, thromboendarterectomy is an important surgical procedure in the treatment of atherosclerosis and its complications.
Thromboarterectomy is one of the most effective ways to treat various types of pathologies in the body’s arterial system, including thrombosis. During this intervention, the surgeon removes the blood clot along with the blood clot that formed it. As a result of removal of the artery at the operated site, in some cases it may be necessary to re-open it.
The most common form of detachment of an atherosclerotic plaque from the vessel wall is the so-called imbibed thrombus. This phenomenon develops in conditions of disturbance of local blood flow due to the location of the plaque in the area of the narrow diameter of the vessel. As a result, when blood circulation is impaired, venous blood forms on its surface (impregnation). As a rule, the formation of a blood clot is always accompanied by the occurrence of an inflammatory reaction in the intima surrounding the vascular plaque, the severity of which significantly depends on the composition and activity of the blood cells present in it.
If thrombosis of the main arteries is detected, it is necessary not to postpone the operation, since the possibility of complications increases every day. At the onset of the disease, the normal blood supply to the organ is disrupted. This is how hypoxia develops, the concentration of toxins in the bloodstream increases, and the immune system is weakened. Subsequently, there is an acute danger of death for patients.
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