Glazobubonic tularemia

Tularaemia occulobubonia is an acute natural focal zoonotic infection, accompanied by specific damage to the conjunctiva and buboes. The pathogen can affect many organs, but most often affects the liver, spleen and lymph nodes. The source of infection is rodents, pets, as well as people sick with various forms of the disease. There are cutaneous, glandular and primary affective forms. The infection is severe and patients may experience complications. The most common eye lesions are conjunctivitis and dacryoadenitis.

Symptoms. Per

Tularemic Finnish eye. Despite the fact that diseases associated with tularemia are extremely rare, patients in the so-called typhoid departments remember him as a pest of peace. This infectious disease most often affects the population of the southern and eastern parts of Russia, but bobby also comes to other regions. And how it comes! Until the summer of 2013, in the Kursk region, foci of tularemia were identified in the Sudzhansky, Cheremisinovsky and Glushkovsky districts. In addition, 7 epidemic foci were discovered during 2008 in the Leninsky district of the Moscow region - in the villages of Volna, Kakelevo, Kryukovo (two foci), Perino, Gorki and Selivanovo.

Diseases of the tularemic Finnish eye, according to doctors, were associated with specific forms of this infection - the so-called oculobubonic tularemia. It does not currently occur due to the decrease in the range of the parasite in the Russian environment and the disappearance of the danger of infection of livestock. But this does not stop the disease from being a horror story for adventure lovers. But tularemia continues to be remembered as an infectious disease. For example, in the book of the famous scientist E.I. Korenberg on parasitology, when describing the clinical picture of the tularemic Finnish eye, elements of brucellosis are mentioned. Okay, but what about those who can’t remember this form of tularemia infection? Textbooks on microbiology and epizootology of domestic animals come to the rescue - where only an interesting detail about this type of human infection is revealed. It turns out that tularemic birds of the weed and carnivore family have sharply protruding buboes on the neck, and they are clearly separated from healthy skin tissue. Scientists explain the presence of buboes by the fact that birds have at their disposal all the necessary knowledge about the characteristics of this infection. Well, as for epidemic outbreaks of tularemia, the eyelids remain drooping, although something else can be learned. For example, some scientists identify the following stages of tularemic Finnish eye. At the first stage, bullshit is thrown in the form of a significant bubo of the hollow neck muscles of calves and lambs. After some time it begins to itch there, then progresses with severe pain and hyperemia. Adult yaruzas suffer especially, as they get such a bubo in several places at once. Moreover, the bubo can block vision. Pain from injury is typical for females and males. There is evidence of the occurrence of a bubonic complication a few days after the administration of drugs against bedbugs. By the way, a similar syndrome sometimes occurs with barley. From the internal organs, tulareca occurs without any difficulties. This is where nature helps. The fact is that the mass extermination of migratory birds, including magpies and crows, reduces the spread of the infection. However, this must be done competently, since a passerine bird may suffer from a blizzard. According to gerontologists, these birds should disappear from the face of the Earth, then the tularemic Finnish eye will

**Tularemia ocular glandular**

Etiology

**Tularemia eye beetle** is a motionless sessile creature that parasitizes the lens, iris, vascular peduncle and tip of the ciliary body of the eye, as well as the vessels and nerve fibers of the bubo itself. Causes a disease - glandular ocular tularemia.\n\nWhen a moving insect gets into the eye, accompanied by a characteristic rustling sound that frightens a person, tularemia occurs at lightning speed - epidemic conjunctivitis, during which the bird and insect die. When the parasite stops moving, the pathogen remains in the victim’s body. An inflammatory process begins, manifested by the formation of bubbles of various sizes in the wall or anterior chamber of the lens. The mass itself gradually becomes cloudy, takes on a yellow or orange tint, the membrane of the vesicles is infiltrated around the mass of the entire iris and the corneoscleral choroidal endings of the vessels. The capsule then becomes overgrown with fringed villi, resulting in the development of a bubo granuloma.