Sideropenic anemia

Anemia sideropenia is one of the most common types of anemia, which is characterized by a lack of iron in the body. This can be caused by a variety of reasons, including insufficient dietary iron intake, blood loss, gastrointestinal disease and other factors.

Symptoms of sideropia anemia may include fatigue, weakness, shortness of breath, pale skin and mucous membranes, as well as headaches and dizziness. If left untreated, anemia can lead to serious health consequences such as heart failure, stroke, and other diseases.

To diagnose siderapy anemia, it is necessary to conduct a blood test and determine the level of hemoglobin and iron. Treatment usually involves taking iron supplements, which help replenish iron deficiency in the body. Dietary changes may also be recommended to increase your iron intake.

It is important to remember that siderapy anemia can be caused by various reasons, so before starting treatment you must consult a doctor for diagnosis and prescribing appropriate treatment.

Anemia Sideremia

Description

Siderative anemia is a pathological condition characterized by a decrease in the concentration of hemoglobin in red blood cells, a violation of their production and (or) a violation of life expectancy and morphological transformation. As a result of a decrease in hemoglobin concentration, the function of red blood cells to transport oxygen to the organs, tissues and systems of the body is impaired. A decrease in hemoglobin concentration can be primary or secondary. Primary anemia is characterized by a decrease in the supply of iron to the body. Iron is an important component of hemoglobin, so disturbances in its metabolism can also lead to the development of anemia. Diseases accompanied by impaired absorption of iron from food can also only occur due to iron excess or deficiency. Secondary anemia is a consequence of the body’s increased need for oxygen during muscle exertion, pregnancy, and pathological processes in the body. The production of erythropoietin, which is induced by kidney disease, leads to increased physiological iron requirements, which contributes to the development of secondary anemia, especially in patients with chronic kidney disease. Additionally, if large amounts of sex hormones are produced, women have an increased need for hemoglobin to increase blood volume during menstruation. This leads to hyporegeneration of iron, even with normal or moderate vitamin C levels or in the absence of anemia for a long time before menopause. Impaired iron absorption in end-stage chronic renal failure may also be accompanied by the development of anemia, regardless of the patient’s age. Excessive output of hemoglobin from