Blockade Peri-infarction

Abstract Blockade can be of several types, and each of them has its own effect on the effectiveness of therapy for myocardial infarction. Intoxication with various chemicals, such as salts of heavy metals, barbiturates, diuretics or thyreostatics, and alcohol abuse can provoke and aggravate the development of vascular dysfunction. Damage to peripheral and coronary vessels leads to a deficiency of oxygen and nutrients in the heart muscle. The appearance of the blockade allows us to assess how bad the patient’s condition is.

Form of attack Blockades have a transit form, and occur in most cases as a transient part of a disturbance in the repolarization of the myocardial ventricles. During an attack, it seems that the pathological process is localized in the myocardium. In time, this condition is observed for 5-8 minutes. After this, the time for natural contraction of the heart muscle passes, then a feeling of a lump arises in the chest, the pain radiates to the arm. Cold sweat also appears. Then the condition returns to normal. Symptoms disappear, the patient feels healthy. For treatment, drugs such as analgesics, anticholinergic drugs, beta-blockers are prescribed. Before starting treatment, you must undergo an ECG. This technique is used to prevent paroxysmal tachycardia. It is performed after an attack, when the symptoms have subsided.

**Blocks in the ventricles** *manifest a clinical picture that resembles left ventricular hypertrophy.* The pathology develops relatively slowly, starting 2 weeks after the attack. There are three types: large, small and normal. Large ones are characterized by two negative waves in the first part of the cardiac cycle on the ventricular ECG waveform or inversion changes in the precordial leads. Small ones are changes in the second part. *Large block:* Distal. The conduction system of the ventricles is affected, suppression of the propagation impulse is observed, with the exception of pathological changes in the coronary bed of the heart. There is no myocardial ischemia in this area. Diffuse. Multiple lesions of the conductive vector are observed, which occurs due to the large volume of the lesion. Transitional. It is focal in nature and precedes ischemia or intoxication. In the absence of negative waves on the electrocardiogram.

Coronary heart disease (CHD), also known as coronary heart disease or non-ischemic cardiomyopathy, is a chronic failure of the myocardial blood supply caused by various disorders of blood flow through the coronary arteries as a result of atherosclerosis. Peripheral conduction circuit blocks are the most common manifestation of coronary artery disease and can be classified according to various anatomical causes. Transmural myocardial infarction is the development of necrosis in the wall (endocardium) of the heart, leading to changes in the electrical activity of the ventricles.

Definition of peri-infarction blockade Transient blockade of the peripheral chain is carried out through the zone of necrosis, which is located to the middle muscle and upper tissue of the affected area, and has the character