Hypertension Renoprivative

The most common cause (80–95% of cases) of the development of renoprival hypertension (RHT) is kidney pathology: glomerulonephritis in the initial stage in the absence or minimal severity of edema and arterial hypertension. In the case of limited damage to the glomeruli in glomerular syndrome and other kidney diseases, its decrease occurs and renoprival hypertension develops. Also a disease associated

The name “renopriv hypertension” has become widely known in the scientific literature for a long time. Professor Bouchaud 35 years ago made a report on the existence of a new type of arterial hypertension. L.A. Verbovensky in 1967, and after him O.S. Radchenko (1971), gave this disease the name “renoprivative disease.”

Renoprial hypertension is a form of renal hypertension that has a specific etiology: hemodynamic impairment occurs with a simultaneous decrease in glomerular filtration and a decrease in the velocity of blood flow above the muscular filter in the renal arterioles. As is known, a decrease in glomerular filtration is most often associated with a narrowing of the afferent arteries of the kidney, and a slowdown in blood flow is possible both due to a narrowing of the blood flow itself and due to a violation of hematocrit. Both mechanisms lead to decreased renal function. Consequently, a decrease in GFR leads to