Rickettsiosis Tick-borne North Asian

NORTH ASIAN TICK-BORNE RICKETSIOSIS

North Asian tick-borne rickettsiosis (syn.: North Asian tick-borne typhoid fever, Birobidzhan tick-borne fever, Siberian tick-borne typhus, Siberian tick-borne typhus, tick-borne typhus, North Asian tick-borne typhus) is an acute infectious disease with a transmissible mechanism of transmission of the pathogen and predominant damage to the vascular system, characterized by fever, intoxication, damage to the heart, blood vessels, and central nervous system.

The causative agent is Rickettsia sibirica from the group of tick-borne spotted fevers. The reservoir and vector are mites of the genus Dermacentor. The disease is common in Siberia, the Far East and Yakutia.

Clinically, mild, moderate and severe forms are distinguished. The incubation period is 5-10 days. The onset is acute, with chills, a sharp rise in temperature to 39-40°C, severe headache, body aches.

Characterized by a hemorrhagic rash on the skin and mucous membranes, possible nosebleeds. Changes in the heart (myocarditis) and blood vessels (endarteritis) are noted. Treatment: tetracycline antibiotics, prevention - vaccine.

North Asian tick-borne rickettsiosis, also known as North Asian tick-borne typhoid fever, is an acute bacterial natural focal vector-borne disease that occurs with high body temperature and symptoms of general intoxication (poisoning), less often with damage to the central nervous system, manifested on the skin as a small-spotted rash and enlarged lymph nodes. The reservoir and source of infection are farm and wild animals (mainly rodents, dogs), and the carrier of the causative agent of zooanthroponotic disease is ticks.[1] Infection occurs when a tick suctions or attacks the skin during blood-sucking, or when the pathogen scratches the affected area.