Rhinitis Vasomotor

What is rhinitis? When you can't breathe... Oxygen surrounds you everywhere. However, due to the imperfection of pulmonary ventilation (ensuring air circulation in the lungs and tissues), its content in tissues and blood leaves much to be desired. There is good reason to believe that a decrease in oxygen concentration in the blood has a very adverse effect on body functions. This is especially true in some clinical situations. Thus, anoxemia (low oxygen content and increased carbon dioxide content in the body) is a serious problem of life. One can imagine situations where oxygen deficiency can reach 20 cm Hg. Art. The conditions that arise during a forced, even hyperventilation, increase in carbon dioxide tension in the blood are of greatest practical importance. The latter has the following effect: an increase in carbon dioxide tension causes an increase in the content of hydrogen ions H+ in the blood, an increase in the CO2 content and is accompanied by a shift in blood pH to the left. At the same time, assimilation processes (tissue respiration) are inhibited, the permeability of tissue cell membranes increases, and tissue metabolism (biological transformation of various organic substances in cells) is inhibited. At the same time, the efficiency of a number of physiological systems decreases. These negative factors grow rapidly and, if their effect continues for a long time, can cause serious disorders of life - a catastrophe for



**Vasomotor rhinitis** (syn. neurovegetative-vascular rhinitis): persistent difficulty in nasal breathing caused by spastic contractions and dilation of the vessels of the nasal cavity. The causes of vasomotor rhinitis are varied. Often this disease is caused by the consequences of a chronic infectious disease of the nasal passages and paranasal sinuses, hypertension, atherosclerosis, diabetes mellitus, migraine, multiple cerebrovascular accidents and some other diseases of the central nervous system. There are cases when the cause cannot be determined. Predisposing factors are considered to be changes in the sensitivity of nerve endings caused by prolonged irritation of the vasomotor centers by chemical agents (including sulfuric acid), drugs or cold when working in hazardous industries. Changes in sensitivity are facilitated by the morphological features of the anatomical structure of the vascular apparatus of the nasal cavity, which manifests itself in the form of difficulty in venous outflow from the nasal vestibule system, etc. Neurasthenia, mental illness, collagenosis, allergic reactions, and vasopathies contribute to the occurrence of clinical manifestations of B. r. as a concomitant disease. A vasomotor reaction of the neuroreflex type often develops in individuals with a neuropathic tendency, a tendency towards hypochondria, and hysteria. With constant negative psychological influence, depleting reserve adaptation mechanisms, the disease can manifest itself. R. of the neurotic type can also develop when the nature of the functioning of the body changes as a result of prolonged overwork, fatigue, disruption of work and rest, rest and sleep, physical and mental activity. Mental unrest, stress, emotional overload, unrest at work in conditions of the need to perform difficult work in breaks between two others should be assessed as provoking factors that can contribute to an increase in the level of emotional stress, the development of neurosis and, ultimately, the emergence of neuroreflex or neurovascular reactions in mucous membrane of the nasal cavity and nasopharynx. Therefore, an invaluable role belongs to the doctor, who not only detects pathology, but also treats the patient so that these functions are performed by one person through a compromise treatment, taking into account an individual approach to the patient, depending on personal habits, affectivity, level of self-awareness, and hereditary burden of heart disease. , blood vessels, neurological disorders from the standpoint of endogenous risk factors for the development of obstructive processes in the nasal cavity (formation vasomotility, persistent changes in hemostasis). The subject of study of this disease is also environmental factors, which often manifest themselves as having a direct etiopathogenetic nature (increased air pollution in large cities, its humidity, humidity