Stirlina Symptom

Stirlin's symptom is a sign that is used in medicine to diagnose diseases of the lungs and chest. It was described by the German surgeon and radiologist E. Stirlin in 1918.

Stirlin's sign occurs when a person coughs or sneezes, exhaling air through the mouth and seeing his mouth fill with liquid. This happens because there is fluid in the lungs that comes out of the airways along with the air.

To diagnose lung disease using Stirlin's sign, it is necessary to perform a chest x-ray and evaluate the presence of fluid in the lungs. If fluid is detected, it may indicate the presence of pneumonia, tuberculosis or other lung diseases.

In general, Stirlin's sign is an important diagnostic tool in medicine, which helps determine the presence of fluid in the lungs and identify lung diseases at an early stage.

Stirlin sign is a Strichlin-Simpson sign, which is named after Heinrich Stridlin, a German radiologist who first described this symptomatic sign in patients with chest trauma in 1886.\nIt is one of the most common symptoms in the clinical neurological examination of spinal cord injuries. When there is a traumatic injury to the spinal cord or spinal root, free scoliosis is formed - a dislocation of the spine. This condition is usually described as "twisting" - a sharp flexion and extension of the spine in the typical position of the patient. In the absence of spinal trauma, spinal dislocation does not occur, because the spinal column does not lose flexibility. This method of injury can lead to compression syndrome, in at least 93% of victims - syringomyelia, syringobulbia, and in 32.4% - compression of the spinal cord (internal tumor of the dorsal radicular cauda equina, venous angiomatosis, proliferation of neurogenic glioma). Symptoms pathognomonic for damage to the anterior spinal rhombus are functional movement disorders. Injuries to the cervical spinal cord are characterized by peculiar autonomic dysfunctions of the sympathetic and parasympathetic centers, which make up structures called, according to Lermitgen’s proposal, three-neuron regulation of autonomic vessels and the heart. These sympathoadrenal disorders support an increase in blood pressure - the so-called sympathoadrenal sympathicotonia, and in the upper extremities this leads to paresis of the extensors of the fingers, arms, muscles of the distal limbs, the development of anhidrosis and the phenomenon of central pinpricks, and the possible development of Lasegue's symptom. Compression of the medial parts of the dorsal rhombus causes a rapid decrease in blood pressure, sometimes orthostatic collapse, a collapsed state. Lesions affecting the lateral rhombus quickly cause manifestations of cachexia, trophic disorders, neurodystrophic autonomic disorders, collapsoid edema, pseudoperitoneum syndrome, and generalized adrenal dysfunction. Important symptoms of damage to the lateral rhombus are disorders such as circulatory failure, hemisyndrome, especially hemiplegia, hyperesthesia, and some syndromes associated with impaired cerebral circulation with the formation of a hematoma.