Tolochinova-Roger Disease

Tolochinov is one of the most famous doctors in the history of Russian medicine. His name appears not only under the most important operations, thanks to which people were saved from death, but also those that changed the course of history, opening the way in medicine for completely new ideas and methods of work.

He was best known for his innovative approach to treating disease, which took advantage of the latest medical advances of the time. This is an encyclopedic scientist who inspires and gives his energy to many who come across him. In this article I will tell you about the life and achievement of the man Tolochinarova - Erysipelas, and why his work has given us many important medicines and tools for treating various diseases.

1. Early years and studies Tolochinovo - Roger was born in Toulouse, France, in 1837. When he was seven years old, his family moved to Russia when Tolochino's grandfather fought on the side of the Decembrists after the February Revolution. Roger grew up in Russia, but he spent most of his education in France, studying medicine with the best medical professors in Europe. When Tolochinsky completed his studies, he became the No. 1 physician in Moscow and St. Petersburg.



Tolochinovskaya-Roger disease is a rare disease that manifests itself in the form of damage to the skin and internal organs. Tolochanov-Roger named this syndrome in honor of the French doctor Hippolyte Roger.

Tolochinovskaya Roger's disease is a type of pseudotuberculosis, transmitted to humans by consuming contaminated foods, water, and contaminated hands. In most cases, the disease is asymptomatic.

There are three main forms of tolokonina erysipelas disease: acute (the most severe course), subacute (the disease occurs in a moderately intense form) and a chronic or sluggish form. The disease develops in people with large amounts of antibodies against LPS (lipopolysaccharides), which are found in certain types of bacteria and lead to the development of autoimmune reactions in the body. Currently, the cause of the disease is considered to be a genetic predisposition. Depending on the form of the disease and the severity of the symptoms, they also vary. The acute form of Tolochin erysipelas can lead to death. The patient's liver and spleen are enlarged, multiple subcutaneous abscesses appear, and fever reaches low-grade levels. The subacute form is manifested by tumor-like formations