Pulmonary tuberculosis Fibrous-cavernous

Pulmonary tuberculosis fibrous-cavernous - i.e. a complicated form of pulmonary tuberculosis, in the pathogenesis of which residual disseminated lesions, sclerosis of the lung tissue, hemorrhages, atelectasis, multiple cavities, fibrosis with severe deformation of the pulmonary pattern, the phenomena of bronchiectasis and bronchosclerosis play an important role. Occurs due to irrational treatment of acute primary and hematogenously acquired tuberculosis. The process is often bilateral, progresses slowly, the outcome is pneumofibrosis, bronchospastic syndrome, amyloidosis. There is practically no damage to the lymph nodes and the osteoarticular system.

Pulmonary tuberculosis is a group of infectious and inflammatory diseases of the human body caused by a specific pathogen - Mycobacterium tuberculosis, accompanied by necrosis of lung tissue, lymph nodes and the formation of pronounced fibrous changes.

The causative agent of the disease is Mycobacterium tuberculosis, which can be transmitted from patients or carriers by airborne droplets. The causative agents of tuberculosis multiply in the lymphoid tissue of the respiratory tract - lymph nodes. As a result, a local tuberculomatous process develops, usually without symptoms. Then mycobacteria penetrate into extrapulmonary organs and tissues. With sufficiently long-term contact with a patient with tuberculosis, mycobacteria invade other human organs, and a generalized form of tuberculosis infection develops. Most often, tuberculosis processes develop in the lungs, against the background of a reduced immune system, long-term infectious lesions, or the presence of a tumor process. The disease can develop against the background of immunosuppression, and