Apophysis Persistent is a pathological condition in which there is constant or periodic retention or growth of anatomical structures such as teeth, bones and cartilage. The term was coined in 1939 by English surgeon James Phillips, who described a case of retained teeth in a man who died of cancer at the age of 60.
Apophysis Persistent can occur in various parts of the body, including teeth, jaws, nose, ears, fingers, lips and bones. It can be caused by various factors such as genetic predisposition, trauma, infections, bone and dental diseases, and other factors.
Symptoms of Apophysis Persistent may include pain, discomfort, changes in the shape and size of the organ, and difficulty chewing, speaking, and breathing. In some cases, Apophysis Persistent can lead to serious complications such as infections, fractures, and organ deformation.
Treatment for Apophysis Persistent may include surgical removal of the organ or parts of it, drug therapy, physical therapy, and other treatments. The choice of treatment method depends on the type and stage of the disease, as well as on the individual characteristics of the patient.
Overall, Apophysis Persistent is a serious condition that can lead to various complications. Therefore, it is important to consult a doctor promptly and begin treatment to avoid serious consequences.
Apophysis Persistent is a rare and little-studied disease in which there is an abnormal thickening or complete absence of the neck of the tooth. This pathology can lead to serious complications such as gum inflammation, tooth loss and even malignant tumors in the jaw area. However, there is still no consensus on the causes of this disease. Some experts believe that this may be due to a genetic predisposition, while others see the reason in defects in the development of soft tissues during the formation of teeth. Environmental factors such as vitamin C deficiency, dental trauma and poor oral hygiene have also been suggested. Thickening of the neck of the tooth may also be due to overuse of certain medications, such as steroids or immunosuppressants. Although no definitive evidence has been found to implicate these drugs in pathology, some authors warn of a possible risk.
Another possible mechanism causing persistent apophthesis is ligament dystrophy, in which the connection between the tooth and the alveolar bone is disrupted. As a result, the tooth bone begins to experience a tensile load that it cannot withstand, and the neck of the tooth shifts towards the permanent dentition tooth, which grows under the main tooth. This condition may begin in childhood and continue to develop into adolescence.
Clinically, persistent apophysis is manifested by unusual changes in the shape of the tooth, its position and the appearance of a secondary dental defect. Affected teeth differ from healthy ones in the thickness and shape of the crown; they often have a disproportionately large conical protrusion on the chewing surface. The normal position of the teeth becomes incorrect, as the tooth takes on an inclined position towards the permanent dentition tooth (bevelling of the front and side surfaces of the tooth). Chewing occurs predominantly on one side. This is due to the fact that the constant force of chewing pressure acting on a highly uneven bite defect causes rapid destruction of the microstructures of dentin and enamel of adjacent