Retinal Degeneration Discoid

Retinal discoid degeneration: Understanding and treatment

Introduction:

Discoid retinal degeneration (also known as discoid macular degeneration or macular degeneration) is a serious eye disease that can lead to loss of central vision. This condition is common in older adults and can have a significant negative effect on their quality of life. In this article we will look at the main aspects of discoid retinal degeneration, its causes, symptoms and available treatments.

Causes of discoid retinal degeneration:

Retinal discoid degeneration is associated with age-related changes in the eyes. It usually develops due to the accumulation of fatty deposits and abnormal protein waste in the macula area, which is in the center of the retina. Under the influence of these changes, the retinal cells responsible for central vision are destroyed.

Symptoms of discoid retinal degeneration:

The main symptom of discoid retinal degeneration is the gradual loss of central vision. Patients may notice objects in the center of their visual field become distorted or disappear, making it difficult to read, drive, and perform other daily tasks. You may also have trouble distinguishing colors and decreased vision clarity.

Diagnosis of discoid retinal degeneration:

Diagnosis of discoid retinal degeneration involves examining the eye and performing special tests such as optical coherence tomography (OCT) and fluorescein angiography. OCT allows doctors to obtain a detailed image of the retina and determine the extent of damage, while fluorescein angiography helps assess the condition of the vessels of the eye and identify possible ischemic changes.

Treatment of discoid retinal degeneration:

There is currently no complete cure for discoid retinal degeneration. However, there are methods that can slow the progression of the disease and improve the quality of life of patients. One such method is the injection of drugs such as anti-VEGF factors, which can reduce vascular permeability and inflammation in the retinal area. Special optical devices may also be recommended to help improve vision and make everyday tasks easier.

Conclusion:

Retinal discoid degeneration is a serious condition that can cause loss of central vision in older adults. Understanding the causes, symptoms, and treatments of this condition is essential to providing quality care to patients. Modern diagnostic methods make it possible to promptly detect discoid retinal degeneration and begin appropriate treatment. In the future, new treatment approaches may be developed that will help manage this disease more effectively. At this time, it is important to seek the help of experienced professionals and follow their recommendations to manage the condition and preserve visual function.

Macular atrophy without disc edema is characteristic of severe stages of retinal degeneration of the Kunt type. It leads to hardening and thickening of the macular region and macula, which ultimately leads to the inability to see color vision and left lesion.

Process Discoid degenerations manifest as multiple, narrow, deep retinal lesions located around the center of the macula. If progression is rapid, concentric discoloration forms from the center to the edge of the macula and causes retinal detachment. With the eccentric location of white shading, which usually occurs with diffuse degeneration at an older age, absorption of macular pigment occurs in the pole area.

Optical techniques include fluorescein, OCT, and optical coherence tomography. Visual field visualization involves a reduction in central perceptual volume or visual interference. Nonspecific signs may include deterioration of visual acuity, narrowing of field boundaries, halo effect and distant background influence.

In the diagnosis of discoid degeneration, the central lesion may be missed until sudden deterioration of the central visual field occurs. However, this is a rare event and significant deterioration results in the loss of the central bell-shaped area of ​​the macula, and finally the loss of the central half. Gradually the paicular depression spreads until the remaining