The main reason for the development of diabetes after surgery is increased blood glucose levels and insulin resistance. Elevated insulin levels can also trigger the development of MDS. Postoperative hyperglycemia causes acute activation of the sympathetic nervous system and, consequently, the release of histamine and adrenaline. This, in turn, leads to
Minor surgical diabetes: a combination of acidosis and hyperglycemia in the postoperative period
In the postoperative period, patients may encounter various complications, including minor surgical diabetes. This condition is characterized by a combination of acidosis and hyperglycemia and can occur after various surgical procedures.
Diabetes minor is a temporary condition and usually develops in individuals with no history of diabetes. However, patients with a predisposition to diabetes or other risk factors, such as obesity or a family history of diabetes, may have an increased risk of developing the condition.
The main mechanism for the development of minor surgical diabetes is associated with physiological stress caused by surgery. During surgery, the body undergoes various stress responses, including activation of the sympathetic nervous system and the release of stress hormones such as cortisol and epinephrine. These physiological changes can cause increased blood glucose levels and impaired sugar regulation.
Hyperglycemia, or elevated blood glucose levels, is one of the main signs of minor surgical diabetes. Elevated glucose levels may be due to a decrease in tissue sensitivity to insulin or an increase in glucose production by the liver. As a result, normal glucose metabolism in the body is disrupted.
Acidosis, another characteristic of minor surgical diabetes, results from the accumulation of ketone bodies in the blood. When glucose levels are high and insulin availability is low, the body begins to use fat instead of glucose as an energy source. This leads to the formation of ketone bodies, which, if their accumulation is not controlled, lead to the development of acidosis.
Symptoms of minor surgical diabetes may include increased thirst, frequent urination, fatigue, dry skin and mucous membranes. In addition, patients may also experience drowsiness, poor concentration, decreased appetite, and weight loss. In some cases, if minor surgical diabetes remains unrecognized and untreated, diabetic ketoacidosis coma may develop, which is a serious complication.
Treatment for minor surgical diabetes usually involves controlling blood glucose levels and normalizing the acid-base balance. Doctors may prescribe insulin to lower glucose levels and control acidosis. In addition, it is important to keep the patient hydrated and ensure adequate fluid intake.
Preventive measures can also be taken to reduce the risk of developing minor surgical diabetes. It is important to review the patient's medical history and determine whether there are risk factors such as diabetes or obesity. If these factors are present, steps can be taken to optimize glucose levels before surgery, for example through lifestyle changes including physical activity and diet.
Minor diabetes is a temporary condition, and with proper treatment and management, most patients make a full recovery. However, it is important to seek medical attention if you experience symptoms or complications after surgery to ensure that minor surgical diabetes is diagnosed and treated promptly.
In conclusion, minor surgical diabetes is a condition that can occur in the postoperative period and is characterized by a combination of acidosis and hyperglycemia. It is caused by physiological stress caused by the operation and is temporary. Early detection and adequate treatment are important aspects of managing this condition and reducing the risk of complications.