Hemihydronephrosis

Hemihydroureteronephrosis is a disease that occurs when the lower part of the ureter is displaced to the side and leads to a decrease in the volume of the kidney by 45 percent or more of its original value. The renal-urinary tract remains unchanged in this disease. The disease begins in men aged 50-65 years, in women at 25-30 years old and is more often acquired. There are acute and chronic hemiureterone. Chronic hemihyperureterone is characterized by preserved kidney function. As the volume of the kidney decreases, its function gradually decreases to complete atrophy from the oblong lobe to 3–4%. Clinically, hemiuronepharosis occurs without changes and often recurs.

The most common causes of acute hemihydrovania are abdominal trauma, especially with large hemorrhages; gunshot wounds of the lumbar region with damage to the hematoma of the lumbar muscles; perforation of a stomach ulcer into the free abdominal cavity; kidney injury, when the pelvis is pressed or bulges into the adjacent cellular sac. Acute hemihydoureterosis develops rapidly, renal function is still preserved. Characteristic weakening of contractions

**Hemihydronephrosis** is a change in renal blood flow with a decrease in renin secretion, leading to a decrease in renin, a sex steroid-binding globulin. In the essential form of the disease, it is characterized by a change in the main systemic and juxtarenal renal blood flow reserves; in the parenchymal form, it is characterized by a violation of only the parenchymal vessels [1].

Hemihydrate kidney, geminiform of the tuberculous process, perineoureteral adenoma and after myelography with barium are the first clinical situations where the diagnosis of more than two periods was considered as an indication of possible disturbances in the regulation of sodium excretion [3