Hypersomnia Hypoglycemic

Hypoglycemic hypersomnias (HH) are a group of sleep disorders characterized by excessive daytime sleepiness, daytime fatigue and weakness, accompanied by night awakenings and a sleep duration of about 8 hours or less. According to European studies, the prevalence of HS in Europe reaches 14%. The disease is based on changes in glucose metabolism in the body. The primary form of the disease is associated with pathology of the sleep center in the brain. Excessive production of serotonin leads to an increased need for sleep. At the same time, chronic fatigue, emotional lability, and weakness occur. In patients with an excess of this group of mediators, the sensitivity of the respiratory center to the neurons of the hunger center decreases. Drowsiness in this case comes by reducing the concentration of glucagon in the blood, which stimulates appetite.

Hyperglycemic hypersomnia is pathogenetically similar to the neurosensory form. The neurons innervating the appendix are affected and as a result of their damage, hypersomnia, drowsiness, intermittent sleep and increased production of somatotropin occur. The clinical picture of the disease usually develops at night. During the day, the patient is alert, has severe thirst, urination is more abundant and frequent, and appetite is reduced. At night, the urge to urinate predominates, and the urine contains sugar. Patients are afraid to fall asleep and stay on their feet as soon as possible. In the morning there is a pronounced feeling of weakness, muscle weakness, and drowsiness. Already in the morning there are complaints of constant sleepiness during the day, muscle weakness and impaired attention. Later, after a few days, sweating, headaches, palpitations, apathy and mood swings, lack of will, depression, and sometimes trembling of the fingers, small or large tremors, and eczema-like rashes occur. Hypersomnia is accompanied by increased irritability