Pylethrombosis

Pylethrombosis: causes, symptoms, diagnosis and treatment

Pylethrombosis is portal vein thrombosis, which is characterized by the formation of a blood clot inside the vein. This is a serious disease that can lead to various complications such as portal hypertension and cirrhosis of the liver.

The causes of pylethrombosis can be different. One of the main causes is umbilical infection in infants. In addition, pylephlebitis, cirrhosis of the liver and some liver tumors can also cause pylethrombosis.

Symptoms of pylethrombosis can vary and depend on how severe the disease is. Some of the most common symptoms include pain in the right upper quadrant of the abdomen, nausea, vomiting, abdominal distension, and yellowish skin and eyes.

Diagnosis of pylethrombosis involves various tests such as ultrasound, CT scan and magnetic resonance imaging. These methods allow doctors to determine whether there is a blood clot inside a vein and assess the extent of liver damage.

Treatment of pilethrombosis depends on the degree of development of the disease. In some cases, surgery may be required to remove the blood clot. In addition, the patient may be prescribed medications to thin the blood and reduce the risk of recurrent thrombosis.

After suffering pilethrombosis, the patient often develops portal hypertension, which is characterized by increased pressure in the portal vein. This condition can lead to various complications such as bleeding from varicose veins of the esophagus and stomach.

In general, pylethrombosis is a serious disease that requires timely diagnosis and treatment. If you notice any symptoms associated with pylethrombosis, contact your doctor for qualified medical care.

Blood clots in the esophagus can lead to a disease such as pyletthrombosis - a blood clot in the abdominal vessels that supply blood to the abdominal cavity. The most common causes are umbilical infections in infants and liver tumors, particularly liver cancer. Treatment for pilethrombovatus should be immediate and includes the use of antibiotics, surgery or hepatoresection (removal of part or all of the liver), followed by removal of the clot through an incision or stoma. With pilethrombate, patients may experience abdominal pain, nausea, vomiting, diarrhea, and decreased appetite. Surgical treatment includes excision of the abscess or tumor lesions, removal of the blood clot, and replacement of blood loss with intravenous infusion of crystalloid or colloid solutions

Pilethrombosis is thrombosis of the common hepatic and (or) portal vessels with the formation of large thrombi (when the vascular patency is blocked by more than 75%) or their obstruction throughout.

Pilethrombosis causes mortality in 83% of cases due to obstruction of blood outflow from the stomach, entry into the venous system of its contents from the small intestinal loop, the proximal part of which is deprived of blood supply, resulting in aseptic inflammation (amyloidosis) of the serous membrane and its infiltration with segmented leukocytes

The main cause of PI development is infection of the umbilical vessels. With the development of phlebitis of the portal inferior vein, the risk of spontaneous recanalization of thrombotic masses, which are widely used abroad, usually without any success in case of recurrence and rethrombosis, increases significantly. Considering the significant technical complexity of EPVP and the risk of complications of this intervention in a number of

Pilethrombosis is the formation of blood clots in the portals of the liver as a result of infection or injury, which leads to the development of portal hypertension and circulatory disorders. Occurs more often in men over 45 years of age. Concomitant diseases are: alcoholism, severe burns, blood stagnation, endocrine disorders, prolonged standing or prolonged lying down. Decompensation of the disease is sudden, even with minor changes in diet. Overcrowded venous pathways are compressed and narrowed, increasing the load on the already dilated hepatic vessels, which upsets the balance in the blood circulation and damages their wall. As it progresses, blood flow to the tissues causing the obstruction increases, edema develops, and signs of acute cardiac, respiratory, and renal failure appear. The patient's blood pressure drops sharply, vomiting fountains, cyanosis, clouding of consciousness, temperature instability appear, breathing rhythm is disturbed and a stroke occurs. Sometimes swelling can spread to the inferior hollow