Thrombosis Parietal

Thrombosis of the walls

The blood in the veins and arteries is never in a stagnant state. It moves through the vessels, compressing due to the elasticity of the vessel walls. With any obstacle, it thickens and clogs the lumen of the vessel. The result of this blockage is blood clots.

The process of thrombus formation occurs in two stages: 1. Formation of a fibrin thread - the protein basis of the thrombus; 2. Growth of a blood clot on a fibrin thread. The formation of a blood clot is a natural process; it helps the body protect itself from damage and obstruction of blood flow. However, often blood clots are swallowed into the arterial bed or transferred from one vein to another, which can lead to serious problems and even life.

Classification Parietal thrombi are divided into floating and non-floating. Inside the artery: * Attached to the intima of the artery wall - this indicates that the thrombus adheres tightly to the artery wall, but passes through its lumen to a greater length. * Interocoronary (precoronary) - attached to the wall of the coronary artery immediately at the point where it passes through the dense bone in the wall of the heart. The thinned walls of such arteries thicken faster and, as a result, lead to a heart attack. This can happen suddenly and lead to the death of part of the heart muscle. In case of disease of the parietal arteries, surgery is most often required.

If the intermediate part of the artery is affected, the blood clot blocks its internal circulation, this can cause circulatory problems in the limb, and sometimes in other parts of the body. Floating thrombosis is dangerous because a blood bubble can block the vein and disrupt its circulation. The limb could have recovered after “switching off” the mural thrombus, if not for the concomitant inflammatory process.

How it is determined whether they are floating or attached depends on the location of the thrombus:

Floating is observed far from the heart (for example, in small-diameter arteries), it floats outside the vessel and can approach the heart, completely blocking the flow of blood to the lower extremities. Its volume may increase over time, worsening the patient's condition. The disease has a high probability of leading to embolic poisoning. The patient may not have any symptoms until a large blood clot causes an embolism in the arteries of the upper or lower extremities. For example, a floating thrombus can form through the vessels of the leg, which then reaches the shoulder and causes paralysis when it enters the nerves of the arm (venous thrombophlebitis). He needs to be taken to the hospital immediately. The floating form is also called arterial thrombophlebitis. It is dangerous both for the health and life of the patient. In comparison, an attached thrombus stands on a stalk and remains attached to the internal surfaces