Dejerine-Klumpke Paralysis

Dejerine-Klumpke palsy (Dejerine-Klumpke syndrome) is a motor paralysis that occurs as a result of damage or dysfunction of the lower part of the brachial nerve (C5-C6). This nerve is responsible for innervating the muscles that are involved in the movement of the arm and hand.

Symptoms of dejerine-clampeke paralysis are weakness and paralysis of the muscles of the shoulder, forearm and hand. Patients may experience difficulty moving their arm, as well as weakness and pain in the shoulder and forearm.

Causes of paralysis can vary, including trauma, infection, tumors, neurological diseases and other factors. Treatment depends on the cause, but may include medication, physical therapy, surgery, or a combination of these methods.

Dejerine–Klumpke Syndrome.

Dejerine–Klumpke syndrome is a rare progressive disease caused by blockage or obliteration of any of the five major nerves passing through the chest: on the right - the intercostal nerve of the interlobar ligament, on the left - the lumbospinal and dorsal branches of the sympathetic trunk. May be unilateral: more common on the right side. It is rare, accounting for about 0.5–1% of all diseases of the nervous system. Belongs to the category of critical conditions, requiring immediate diagnosis and treatment in a specialized department. The prevalence of DKS is about 20 cases per 1 million population. It occurs more often in men aged 40 to 60 years. Due to compression of the spinal cord, the lumens of the blood vessels narrow, and the vital centers of the medulla oblongata, cerebellum, and midbrain suffer. Cerebral ischemia develops if the vertebral artery is also blocked, with all the ensuing symptoms in the form of severe neuropathology, partial cerebellar agenesis and ataxic phenomena of various levels. Involvement of the sensory nerve root usually excludes other neuromuscular syndromes in the presence of pain and damage or weak-born tissues. The disease begins acutely with manifestations of cerebral disorders. The outcome of DKS may develop myeloradicomyelosclerosis, conformational lesion of the spinal cord, or pressure ulcer of the spinal thickening with local necrosis and cell atrophy. Taking into account the condition after surgery, vascular, symptomatic, and antihypoxic therapy is prescribed during the rehabilitation period.