Hyperemia

Hyperemia

Hyperemia is a symptomatic sign that manifests itself in the form of redness of the skin, mucous membranes, superficial tissues and internal organs. This is the result of an increase in the amount of blood in the vascular bed due to vasodilation due to various reasons. Hyperemic syndrome is one of those that occurs during an inflammatory process in the body, as well as with injuries and bruises of soft tissues. This syndrome is considered not only a symptom, but also an indicator of the presence of a number of pathologies and diseases. **Causes of development****Hyperemia** Varicose veins in chronic venous insufficiency. Venous skin hyperemia occurs due to impaired blood microcirculation during venous stagnation, which serves as a prerequisite for the occurrence of capillary disorders. The reverse flow of blood is prevented by valves connecting large and medium-sized veins. The veins in front of the mouths are dilated. Blood stagnation in all subsequent veins of the upper limb is due to the compensatory reaction of the valve apparatus, consisting of a complex system of valves. Visceral (internal) vessels disrupt blood circulation and cause symptoms called tissue hyperemia; This medical term means redness or hyperemia. Tissue hyperemia, in turn, is divided into angiogenic and hemorrhagic depending on the cause that caused its occurrence and the method of its occurrence. Hemorrhagic hyperemia occurs due to the release of blood from small capillaries into the interstitial space. Tissue or angiogenic hyperemia naturally appears when the outflow of blood through the arteries is difficult. Tissues, together with blood capillaries, deformed by congestion, respond by increasing local blood supply: blood flow through the capillaries increases, blood pressure decreases, and the distal part of the vessel expands; increased local, distal blood supply causes local tissue hyperemia. The consequences that unproductive hyperemic tissue inflammation entails depend on the latter’s ability to include compensatory mechanisms that ensure restoration of the lumen of impaired arterial capillary blood flow. Based on the dynamics and final result (regression, persistent well-being) of tissue inflammation, the following types of circulatory dystrophy are distinguished: reversible or reversible tissue homodynamic, angiographic, combined circulatory disorders associated with temporary arterial hyperemia, persistent circulatory, angiographic disorders. The latter occur in two forms: exudation and infiltration. These include histiocytic tissue non-inflammatory accompanied by cell proliferation, for example, sufficient blood microcirculation is necessary for their normal differentiation; they are also classified as exudative tissue hyperemia of an inflammatory nature, which can be distinguished from other exudative tissue disorders by research aimed at identifying etiological factors and differential diagnosis of hyperemia based on clinical and laboratory data. Symptoms The manifestations of this syndrome largely depend on what etiology led to its occurrence. In case this is it