Sclerodactyly

Sclerodactyly: symptoms, causes and treatment

Sclerodactylia is a pathological condition in which the skin on the fingers becomes hard and immobile. The name of this disease comes from the Greek words “sclero” (hard) and “dactylos” (finger).

Symptoms of sclerodactyly include hard skin on the fingers, thickening of the skin on the fingertips, narrowing of the fingers, and numbness and tingling sensations. The skin on the fingers may also become pale or bluish due to poor circulation.

Sclerodactyly is one of the symptoms of systemic scleroderma, a rare connective tissue disease that can affect the skin, blood vessels, internal organs and bone tissue. However, sclerodactyly can also be caused by other causes, such as Raynaud's syndrome, autoimmune diseases, injury, or chemical exposure.

Treatment for sclerodactyly aims to improve blood circulation, soften the skin, and prevent further thickening and narrowing of the fingers. Topical medications such as creams, ointments and lotions can be used, as well as physical therapy techniques such as massage and exercise.

If sclerodactyly is caused by systemic scleroderma, treatment will be aimed at controlling the condition of the disease as a whole. In some cases, the use of immunosuppressive therapy or hormones may be required.

Overall, sclerodactyly is a serious condition that can impair finger function and limit quality of life. However, early treatment can help prevent further deterioration of the condition and preserve finger function. If symptoms of sclerodactyly appear, you should consult a doctor for diagnosis and treatment.



Sclerodactenia. For many, this disease will be an unfamiliar word because it is rarely found among the population. But if you have already seen this strange squiggle on your hand, then you are faced with sclerodacty.

Sclerodactic is a rare hereditary disease. At its core, the sclerodist membrane is no different from normal tissue. The frame located under the skin turns out to be altered and abnormal. Cells degenerate, change, and new ones are not formed. This entails drying out and subsequent frailty of the skin and becomes life-threatening due to the risk of vascular damage and the occurrence of phlebitis and thrombophlebitis. Hence, by the way, the pigmentation that occurs during illness. The reasons for the spread of this anomaly are still not fully understood. There are debates about what exactly is the legacy