Symptom Subclavian

The Subclavian symptom is characterized by the absence of a pulse in the upper limb and is a sign of occlusion of the subclavian artery distal to the origin of its main branches.

With this symptom, due to blocking of the lumen of the subclavian artery, the blood supply to the upper limb is disrupted, which leads to the disappearance of the pulse in the arm. The causes of occlusion may be atherosclerosis, thrombosis, trauma and other factors that cause narrowing or complete blockage of the subclavian artery.

Diagnosis of the Subclavian symptom is based on identifying the absence of a pulse in the radial artery on the affected side. To confirm the diagnosis and clarify the localization of occlusion, ultrasound duplex scanning of blood vessels and/or angiography is performed.

Treatment consists of restoring blood flow through the subclavian artery, most often through endovascular interventions or open surgery. With timely treatment, the prognosis is favorable.

The subclavian artery sign is a diagnostic clinical sign that is used to determine the presence of pathology or abnormality in the subclavian arteries. Below is a description and causes of this symptom.

101. Subclavian sign Symptom A symptom is a symptom determined by the absence of a pulse in the arm, i.e. absence of pulse pressure, both on the shoulder or forearm, and at the bottom of the pits. It is not uncommon to have a common wrist pulse that 1% of adults do not have. Systolic blood pressure even reaches or slightly exceeds the upper limit of normal. Such a carotid artery is not capable of maintaining hemodynamics. High sphincter function tests and peripheral venous constriction are what cause pulselessness. Symptoms of brachiocephalic syndrome The clinical picture is very similar to fever with nosebleeds. The heart does not work well, and pulsating vascular pressure is maintained only by deep muscle activity, excluding changes in the arms. Cyanosis of the nasolabial triangle is also characteristic; if the arterial pulse begins to disappear, it actively simulates hyperemia, especially in the area of ​​the conjunctiva and cornea. Progressive pallor of the skin, developing against the background of heart failure