Sore throat Herpetic

Sore throat (acute tonsillitis) is an infectious disease characterized by a syndrome of acute inflammation of one or more palatine tonsils (most often the palatine tonsils, but the lingual part of the larynx can also be involved), manifested by an increase in body temperature, moderate sore throat, symptoms of general intoxication, catarrhal symptoms stomatitis.

Sore throat is the name given to many different acute inflammatory diseases of the throat: tonsils (palatine and laryngeal), pharyngitis and peripharyngeal abscesses. In a broader sense, sore throat also refers to acute inflammation of the pharynx, pharynx, larynx, and epiglottitis, so the term is often used to mean “any sore throat” or “bacterial sore throat.”

Etiology of angina. The causative agents of sore throats belong to bacteria (especially streptococci and staphylococci). Less commonly, it is caused by chlamydia, gonococci, diplococci, and herpes viruses. It is most often caused by viruses (mainly Coxsackie viruses), as well as microbes (streptococci, staphylococci, etc.) or the mixed influence of those and other factors.

The cause of acute tonsillitis (tonsillitis) is various infections of the pharynx - both bacterial and viral. The etiological factors of acute tonsillopharyngitis should be divided into exogenous and endogenous. The former include predominantly bacterial (digestive and respiratory intestinal microorganisms, Haemophilus influenzae, pneumococci) and partially some viruses, fungal infections (oral amoeba, candida), dermatophages from coccobacilli and spirochetes. A predisposing factor is active carriage of hemolytic streptococcus. The endogenous factors of acute tonsillitis are primarily the viral (Epstein-Barr virus) and allergic nature of acute pharyngo-tracheitis. Recent studies do not confirm the role of fungal infections (oral amoeba, candida) in the etiology of ENT infections

The pathogenesis of tonsillitis is associated with the action of a damaging factor, which on the one hand caused a reaction of reticuloendothelial macrophages and T-lymphocytes. As a result, alteration of the vascular endothelium and hypercoagulation develop. The latter give rise to a persistent inflammatory background, which requires combating it through nonspecific compensatory and specific factors. It is the latter that are responsible for local and systemic disorders of homeostasis and resistance of the body. Locally, these disorders consist of limiting the outflow of lymph and blood flow. The development of the inflammatory process leads to the development of sensitization of the body under the influence of migrating pyrogens. In patients with tonsillitis, certain pathogenetic links are observed, for example, antigen - antibody. And even with disproportions due to defects in the immune system, the pathological process gets out of control, intensifies on its own, without permission against the background