Rusakova Axillary Bone Resorption

The study of bone diseases is an important area of ​​medical science. One of these diseases is Rusakov’s axillary bone resorption, which was first described by A.V. Rusakov.

Rusakova sinus bone resorption (also known as resorptive type of osteomyelitis) is a rare disease that is characterized by accelerated destruction of bone tissue in the maxillary sinus area. This can lead to serious consequences, such as disruption of dental form and function, as well as cosmetic defects.

The first signs of the disease may appear in the form of pain in the teeth and gums, swelling and purulent discharge from the nose. Later, purulent sinusitis and fistula formation may develop.

The causes of axillary bone resorption are not fully understood, but there are several factors that may contribute to its development, including infectious diseases, trauma, genetic disorders, and long-term use of certain medications.

To diagnose the disease, X-ray and computed tomography studies are necessary. Treatment for axillary bone resorption involves surgical removal of the affected tissue, as well as the use of antibiotics and anti-inflammatory drugs.

Despite the fact that Rusakov axillary bone resorption is a rare disease, its diagnosis and treatment require special attention from doctors and patients. Regular dental checkups and proper oral care can help reduce your risk of developing this condition.

Rusakov axillary bone resorption (a. s. rusakov)1

During atrophic processes, **resorbing bone** is replaced by a new one, which leads to the formation of cavities (cysts) limited by hyaline and papillary bone substance. After removal of the cysts, extensive defective areas of bone tissue consisting of newly formed bone were discovered. A spongy layer forms at the site of the defect. Therefore, it can be assumed that the appearance of rudimentary bone during atrophy is not a consequence of the linear growth of bone substance, but represents the growth of first cancellous and then lamellar bone in the form of an extrarenal generative replacement of resorbed bone [3]. Many examples of bone regeneration during pathological processes associated with their erosion or degeneration have been described. The existence of genesectopia (formation of resorbitol in the form of more advanced antagonist tissues) can be explained by the presence in these cells of specific mechanisms, in particular, inductive regulatory potential