Dorsal Horn Syndrome

Posterior horn syndrome (syndrome cornus pos-terioris medullae spinalis, CPSM) is a rare neurological disorder characterized by dysfunction of the dorsal horn of the spinal cord. The dorsal horn of the spinal cord is responsible for transmitting signals from the spinal cord to the brain, as well as for regulating muscle tone.

Symptoms of CPSM may include weakness in the legs, loss of coordination, decreased sensation in the legs and arms, difficulty walking, and other neurological problems.

The causes of CPSM are not fully understood, but it is believed that the disease may be associated with damage to the dorsal horn of the spinal cord due to injury, infection, tumor, or other diseases.

Diagnosis of CPSM includes a neurological examination, electromyography, MRI or CT scan of the spinal cord and other tests. Treatment for CPSM may include drug therapy, surgery, or a combination of these methods.

Dorsal horn syndrome is a rare but serious condition that can lead to serious health consequences. Therefore, it is important to diagnose CPSM early and begin treatment to prevent complications from developing.

Dorsal horn syndrome is one of the rare congenital diseases, accompanied by damage to the external dural sac and the posterior horn of the cerebellum located on it. As a result of its violation, the pressure of the cerebrospinal fluid decreases. The disease also affects the muscles individually and blocks the function of voluntary capture.

In the clinical picture, dorsal horn syndrome is a disease with a favorable outcome, thanks to the compensatory capabilities of the body.

An important role in the mechanism of development of the syndrome is played by thickening of the middle part of the anterior cerebral sides and a decrease in the height of the structures of the posterior cranial fossa. This pathology can be either isolated or combined with symptoms indicating other diseases of the spinal and cerebral regions, congenital anomalies, and injuries to the cranial brain. Also, patients with the disease may experience disruptions in the functioning of various brain systems. At the same time, an increase in pressure indicators is observed, as well as disturbances in the circulation of cerebrospinal fluid and a decrease in its resorption. These consequences lead to paresis of the intestines, stomach and spinal muscles.