Acetylcholine shock

ACETYLCHOLINE SHOCK

In 1869, the substances responsible for the development of pronounced neurotoxic symptoms in humans and animals in cases of poisoning with organophosphorus compounds (OPCs) were simultaneously and independently named as acetylcholine. FOS are not used in medical practice, but their natural analogues, such as the nerve impulse blockers D-tubocurarine and D-galantamine, respectively, are drugs. To date, scientific interest in the study of FOS and their derivative drugs continues. Until now, the following questions remain unclear: what form of FOS is considered the most toxic for the body, what reactions it causes in the receptors of the electrophysiological structures of the central nervous system, how this effect relates to a particular toxic dose of FOS. If by focal toxicity we understand the ability of a substance to selectively affect limited areas of the brain, then the most pronounced focal toxicity is observed in a number of substances, which include such “medicinal” anticholinergic drugs as tubocurarine, curare, neostigmine (ederidine) . The only exception is ditilin, which, by the nature of its actions on the central nervous system

Acetylcholine shock is a disease in which a drug changes a person’s consciousness, skin color, heart rate and respiratory rate. This phenomenon was noticed after studying schizophrenia and drug treatment for the mental disorder. For the first time, such an attack was discovered in the laboratory of the Bekhterev Institute. Since then, humanity has been diligently trying to understand the mechanism of its occurrence. Bromides cause not only chills, but also the manifestation of certain pathological signs. Acetylated lecithin is part of the structure of acetylcysteine ​​acid. After its administration by intravenous injection, the patient experiences a state of short loss of consciousness, convulsive twitching of the limbs

Introduction

Acetylcholine shock is a neurological complication that occurs during the treatment of schizophrenia by intravenous administration of acetylcholine chloride drugs. This is due to the drug's effect on the neurotransmitters of the body's nervous system, in particular the acetyl chloride cholinergic receptors in the central nervous system. Aceticloin shock is an emergency that requires immediate medical attention.

Etymology

The name "acetycholine shock" describes the neurological changes that occur as a result of the use of intravenous aceticlide chloride to treat schizophrenia. The chemical name of Acetyliclitium Chloride and Anti Deamidase is Acetyliclitium Chloride. Preparations Acetylcholine Cholinergic receptor for the drug Acetylcholine. Physiology