Encephalitis Vilyuisky

**Viluya encephalitis** is an infectious brain disease caused by a virus of the Flaviviridae family and has a tendency to self-infect. Unlike other encephalitis, the Viluya encephalitis virus enters the human brain through blood through transfusion or organ transplantation. This is a disease

**Wiel's encephalitis** - acute tick-borne viral encephalitis. The causative agent is a virus of the enterovirus family. The reservoir and carrier of the virus are rodents, wild predators, less often domestic animals and people, mainly Baikal herring, moose and other animals. The carriers of the virus are ticks. The infection occurs in the forest. Natural foci of the virus exist in the Primorsky-Khabarovsk Territory, in the south of Eastern Siberia (Buryat Autonomous Soviet Socialist Republic, Chita Region, Krasnoyarsk and Irkutsk Territories).

Epidemiology. In some cases, a tick bite can infect a person, who can then become infectious and dangerous for the entire tick season or up to 3-4 months after it. More often the disease occurs in summer and even early autumn. In epidemic-type foci, outbreaks of epidemics occur at intervals of 2-3 years. Sporadic cases of encephalitis are also recorded. The infection is not transmitted to surrounding ticks of a sick person. The role of ticks in the formation of encephalitis foci is unclear. The virus is released from the body of infected ticks only for 7-15 days (individual animals excrete the virus for 30-50 days). The more ticks a person gets, the higher the risk of disease. Horizontal transmission of infection through blood-sucking insects also occurs. The virus is widespread in nature. It lives in the body of the beaver, house mouse, street fly, sea animals, and Hymenoptera (sparrows, marmots, arctic foxes, jays, tits). Curulens uses the blood of rodents, ticks, and sometimes insects as a food source. The epidemic process of a focal type develops according to the type of exo-epizootic. After receiving the vaccine, the incidence of disease is quite low, so the presence of an outbreak is considered to have been disrupted. After the epidemic ends, the disease reappears as the virus is transmitted by rodents into nature from people. With the onset of cold weather, its activity decreases and a long “winter period” begins. Acutely febrile patients dominate the epidemic focus; asymptomatic carriage is extremely rare. The duration of the incubation period is on average 3 days, but in some cases it is 5-7 days or more. Usually the disease develops very rapidly; the least dangerous cases are a gradual increase in symptoms with only flu-like symptoms remaining. The severity of the disease depends on the method of infection and the infecting dose. The most favorable form of manifestation of the disease is described using the example of a child’s disease. Before going to the forest on May 21, he was already sick with ARVI, and he was vaccinated against polio. Coming out to the edge of the forest, the boy felt bad at the moment when a tick was sitting on his hat. Already on May 23, all signs of encephalitis appeared, although the incubation period was considered over. The next day, a single tortuous vessel was identified at the site of infection. This meant the development of reactive vascular inflammation (the most typical