Endocapillaritis

Endocapillary vascular disease 1. Introduction Endocapillary vascular disease is a chronic disease with relapses and remissions caused by thrombosis or stenosis of arterioles and small veins, leading to tissue ischemia. The pathogenesis of the disease does not develop in one day, but over months or years and largely depends on the triggering factor or cause, for example, trauma, endocrine disease, smoking, toxic damage, metabolic disorders, as well as tumor processes or autoimmune diseases. Since the picture of clinical manifestations varies greatly depending on the duration of existing circulatory disorders, the presence of physical disorders in the form of Raynaud's syndrome, osteochondrosis of the cervical or lumbar spine, mycosis, psoriasis, atony of the mucous membrane of the rectum or bladder, in science there is a need to differentiate them according to etiology, pathogenesis, prognosis. The identification of patients with the “acute form” of endocapillary disease became possible thanks to the use of new invasive research methods - ultrasonography and endovascular surgery. With adequate therapy, it is possible to relieve pain for a long time. 2. Etiology and pathogenesis Pathological development occurs for many reasons in the endocapillary channels. Provoking factors include inflammatory processes in the oral cavity, such as gingivitis (inflammation of the gums), stomatitis, which can lead to vasoconstriction and impaired blood flow. Sharp chemicals (acids, alkalis) cause damage to the capillaries of the mucous membrane and deep ulcers, and blood clots form. These same substances can provoke allergies in the gums and blood vessels. In addition, the causes include the effects of poisons and radiation attacks, HIV infection and some bacterial infections, and insect bites. Regardless of who became the causative factor, capillary damage may proceed according to a different scenario. On the one hand, aneurysmal transformation develops; due to the expansion of the lumen of the vessel, the membrane is damaged with the formation of microaneurysms with platelet deposition, but the peripheral blood flow continues to move, thereby supplying the body with nutrients. On the other hand, there is approx.