Cholecystokinin

Cholecystokinin is a hormone that is produced by the cells of the mucous membrane of the duodenum in response to the entry of chyme containing fats into it from the stomach.

The main functions of cholecystokinin:

1. Causes contraction of the gallbladder and release of bile into the intestines. Bile is necessary for the emulsification and absorption of fats in the intestines.

2. Stimulates the production of digestive enzymes by acinar cells of the pancreas. These enzymes are also involved in the digestion of fats.

3. Participates in the regulation of satiety and appetite.

Thus, cholecystokinin plays an important role in digestion, stimulating the processes necessary for the digestion and absorption of fats in the intestines after eating a fatty meal.

Cholecystokinin (CCK) is a hormone consisting of 39 amino acid residues that is secreted by the cells of the duodenal mucosa.

The cholecystokinemic system is a neuroendocrine system consisting of hepatopancreatic neurons and their dendrites that secrete cholecystokinin and other polypeptide hormones. In turn, cholecystokinins are released into the duodenum and stimulate the production of enzymes by the pancreas and contraction of the gallbladder.

The release of bile into the duodenum stimulates the secretion of pancreatic enzymes, which promotes the digestion of fats. In addition, cholecystokinins can stimulate gallbladder contraction, which helps speed up the digestion process and reduce stress on the pancreas.

In addition, cholecystokinins are also involved in the regulation of appetite and metabolism, and also affect the emotional state of a person.

Thus, the cholecystokinetic system plays an important role in the regulation of digestion and metabolism.

Cholecytokine or cholecystokine is a hormone produced by the endocrine cell of the duodenal mucosa. The name itself means “gastrin-like hormone”, since its chemical structure is somatostatin. This type of tissue is closest to the intestines.

Cell organelles - the apical part, which is concentrated in a state of rest. After a food stimulus in the pancreatic, sphingosine controls the secretion of mucus from apicocytes; others remain in a resting state with a flat membrane. New apicocytes without a cytoskeleton on the distal part of the globe begin to synthesize the hormone. What is somatoliberin? It is a hormone regulator. Once released into the bloodstream, the signal increases the level of agonists in the mucous membrane of the duodenum and jejunum. In simple words, when high-fat foods enter the oral cavity, they stimulate the activity of duodenal epithelial cells. The alkali enters the mucous membrane, and the cells increase the secretion of HCl due to intracytoplasmic disks. The effect on plasma membranes transforms somatostatin into the “second phase of ℅ssies”, where it itself already affects the receptors. The receptor is similar to antibodies fixed on the sheath of a nerve or neuron. Thus, it influences the formation of sympathetic innervation. Under the influence of SSC, contraction occurs due to the action on the contractile apparatus of the cavity of the common bile duct. In parallel with this, bile moves into the lumen. Here you can consider the processes of stimulating the movement of the food bolus into the duodenum. This ensures a physical effect on the cavity and glands to produce enzymes. The hormone is not used to influence another organ, since it does not have a negative effect on it. The exception is inflammatory diseases that provoke a lesion from the blood or inner lining. Then the active parts of the hormone pause interact with the acinus. Chemically, the body separates enzymes from the proteins that connect them. It is important to note that cholecytocin only works when there is an accumulation of fat in the body. Somatostatin, released by choleparic myeloma, is able to neutralize the negative effects of the “precursor”. The use of mCT2-antisomanatine is considered ineffective for inflammation of the gallbladder, since the receptors “bring” both mediators to themselves.