Leriche-Esdeca Syndrome

Lerichy-Esdeky Syndrome is a rare disease characterized by inflammation of the nerves in the lower extremities. This syndrome is named after the names of two French and German surgeons - Lerich and Sudak, respectively.

The origin of the syndrome and its symptoms is associated with decreased blood flow in the legs due to narrowing of the arteries that supply blood to the limbs. When the arteries lose their ability to carry sufficient oxygen and nutrients to the tissues of the lower extremities, inflammation of the nerves occurs. This disease is diagnosed only when there are no obvious causes of pain in the legs.

Treatment for Leriche-Esdequi syndrome involves a variety of methods, including physical therapy, medications, and surgery. With early detection and treatment, it is possible to reduce the likelihood of complications and improve the patient's quality of life.

Exercise such as walking, yoga, running and swimming can help improve blood circulation in the legs and reduce the risk of developing Leriche Esdec Syndrome. However, it is important to follow a proper exercise regimen and consult a doctor before starting exercise.

Drug therapy may be aimed at eliminating the cause of the syndrome, such as widening the arteries or reducing blood pressure. Anti-inflammatory drugs are also used to reduce inflammation in the legs, but should not be overused

Leriche-Esdeca syndrome (LES)

The first detailed description of the LES belongs to Jelliner and Eulenburg. In 1942, Ganong proposed a new classification of IHD that included LES. The condition, previously unrecognized or underestimated by clinicians, did not manifest itself in full either during exercise testing or at rest. Later, LES was included among the angiocardiological syndromes of coronary artery disease. The term was coined by Goldberg. Subsequently, the various stages of this syndrome were actively studied by many authors, as a result of which the idea was formed that the branches of the left coronary artery are predominantly affected: the left anterior and, most often, the posterior small arteries of the heart. The term “LES” appeared mainly due to the fact that with a decrease in blood flow through the coronary arteries, increased demands begin to be placed on the heart muscle. Previously, we talked about type II(B) coronary spasm syndrome, which was characterized by insufficient contractility of the left ventricular myocardium, partially observed angina pectoris and severe fatigue at rest or during normal physical activity. It is now believed that LES is accompanied by the same initial predominance of stenosis of the anterior descending branch of the left main coronary artery and the disappearance of T-type symptoms (angina), but in the case of damage to the right coronary artery, the 3rd hypoxic stage develops, which clinically manifests itself as an attack of angina with rapid pain resolution, high myoglobin content in the blood and increased radioactive isotope of the muscle tissue of the heart when studying its distribution throughout the body. In such situations, the ECG records normal T waves or negative T waves in leads I, II, avF and often a discordant decrease in the content of the IGF glycosylate complex in the blood (sim