Insertional mutation

An insertional mutation is a type of genetic mutation in which a new genetic sequence is inserted into the DNA of a cell or organism. This type of mutation can be caused by various factors such as radiation, chemicals or viruses.

An insertional mutation changes the structure of DNA, which can lead to various diseases such as cancer, hereditary diseases and others. However, not all insertion mutations are harmful. For example, some mutations may be beneficial to evolution if they help the organism adapt to its environment.

In order to identify an insertional mutation, it is necessary to conduct a genetic analysis that will determine the presence and location of the new DNA sequence. This can be done using DNA sequencing or other molecular biology techniques.

Overall, insertion mutations are a complex and interesting area of ​​genetics and biology that continues to be studied and developed.

Insertion mutations are the insertion or addition of one or more genes (transposons) in a cell's genome. Insertions are much more common than any other type of mutation in genomes for several reasons. First, most of the DNA within chromosomes is non-coding, and therefore changes in this region are much less disruptive. Secondly, since transcriptomics and gene expression are controlled randomly, some random insertions or extra sequences are in principle harmless, hence from generation to generation. A third factor promoting insertions is that shared DNA between different genomes of organisms may allow transposable elements to cross species barriers and even spread into interspecies lineages such as humans/mouse, in which insertions have been found that are 4 Mb in length across 200 generations [1]. Because these insertions do not show ancestral consequences, they may have become more common during the evolutionary process of the human species. The accelerated spread of these hypermutable elements corresponds to the relatively accelerated genetic drift of these regions of the human genome compared with the smaller number of mutations that arise over time at other sites in human genetic history. There is currently no generally accepted genetic component for this rapid hypermutational growth of transposable elements in the human genome. Such massive in