Syndrome Catatonic Parakinetic

Catatonic-parakinetic syndrome (V.P. Protasov) is a phylogenetically new dysontogenetic process within the framework of pathology, in which throughout its development - at the stage of ontogenesis and phylogeny - the phenomenon under study retains highly hereditary variability and, therefore, there is only a gradual correction of its functional characteristics. Both syndromes represent a degenerate form of suffering, characterized by characteristics that only make it easier to accurately diagnose each of them. In both cases, the first step is to exclude a congenital disabling pathology that causes movement disorders.

Catatonia parakinesis syndrome is a rare mental disorder that manifests itself in the form of catatonic symptoms. Catatonia includes various manifestations such as stupor, negativism, automatism and mannerisms. Parakinetic Catatonic syndrome manifests itself in the form of parakinesis, which occurs without connection with external factors. Treatment of this syndrome can be complex and requires an individual approach. In this article we will look at the concept of Parakinastic Catatonic syndrome, its symptoms, diagnosis and treatment.

Catatonic Parakinetic Syndrome is a mental illness that can manifest itself differently in

Catatonic parakinesis syndrome (Pan-Arumosandi syndrome, Raimondi-Kahan syndrome or disease) is a rare mental illness associated with catatonic agitation and rarely encountered in the population.

Historically, the syndrome was named after the Scottish psychiatrist George Raimondo, who wrote a paper on catatonia in 1928, and the disease was later named after the Greek physician Constantine Kehan, who also described it in 1865. Today the term is still used by scientists to refer to this disease.

Symptoms of catatonic-parakinetic syndrome are characterized by alternating periods of excitation and inhibition, when the patient's behavior changes depending on the phase of the disease. During the agitation phase, the patient may exhibit uncontrolled movements of the limbs, speech may be fragmentary or absent. During the inhibited phase, the patient becomes apathetic or lethargic and often sleeps or appears to be asleep. Also sometimes