Addison S Disease

Addison's Disease

Addison's disease is an endocrine disorder characterized by insufficient production of corticosteroid hormones by the adrenal glands.

Causes

Addison's disease often develops as a result of damage to the adrenal glands by tuberculosis. Also causes may be autoimmune diseases, tumors, injuries, infections.

Symptoms

The main symptoms of Addison's disease:

1. Weakness, lethargy, fatigue

2. Hypotension

3. Darkening of the skin (hyperpigmentation)

4. Nausea, vomiting

5. Decreased appetite, weight loss

6. Pain in muscles and joints

Treatment

Previously, Addison's disease was considered incurable. Currently, the main treatment method is hormone replacement therapy with hydrocortisone or prednisolone. This allows you to compensate for the lack of adrenal hormones and control the symptoms of the disease. With timely treatment, the prognosis for patients with Addison's disease is generally favorable.

Addison's Disease

Addison's disease is characterized by insufficient secretion of corticosteroid hormones by the adrenal glands. Sometimes this disease occurs as a result of a person becoming infected with tuberculosis.

The main symptoms of Addison's disease:

1. Weakness and lethargy
2. Hypotension
3. The appearance of dark spots on the skin

Previously, this disease was considered incurable. However, Addison's disease is now treatable with hormone replacement therapy. This helps control symptoms and prevent the development of life-threatening complications.

Addison's disease, also known as Addison's insufficiency syndrome, is an exo- or autoimmune disease with a chronic course, the causes of which are the deficiency or absence of synthesis and secretion of cortisol and androgens from the adrenal cortex tissue. The disease is characterized by chronic arterial hypotension (usually moderate), hyperpigmentation of the skin and internal organs, amenorrhea in women and impotence in men.

Medical research up to the beginning of the 20th century led to the formation of a fairly convincing opinion that in hypopituitarism, compensatory secretion of corti