Endocarditis Fibrous

**Phimotic endocarditis** - represents the presence of a special form of endobronchial inflammatory lesion. What is such a pathology as endocardial fibrosis? This disease is characterized by the presence of such features as degenerative changes in the wall of the heart, in which the integrity of the walls of the heart is disrupted. As a result of the fibrotic process, a special tissue is formed - endocardial tissue. When it is formed, the size of the holes between the chambers decreases. Initially, an inflammatory process occurs in the valves of blood vessels and heart chambers. Various injuries and chronic pathologies of the body also play a special role. Endocardial fibrosis can occur, for example, due to heart tumors or heart failure. And in addition to this, necrosis of small cardiac vessels also occurs, which is often complicated by the occurrence of an aneurysm of the abdominal cavity and hydropericardium. The presence of the above phenomena enhances inflammatory changes in the endocardium, which simultaneously affect a large number of cardiac chambers and vessels.

In this case, a special role is played by the presence of a disease such as rheumatism of the heart, as well as inflammation of the heart walls, which occurs as a result of the introduction of microorganisms inside. For example, it could be a staph infection. The cause of this non-infectious form of pathology is hypertrophy or stretching in the presence of inflammation of the heart. Damage to the walls occurs due to vascular disorders. Such a process can develop, for example, due to inflammatory changes in the myocardium and thrombophlebitis of the portal vein of the heart, located near the intracardiac valves, and this leads to the formation of fibrin in the vessels. In addition, risk factors for the development of this type of endocarditis include the onset of age-related changes and special heredity. Vascular consequences can also occur with Marfans syndrome, which manifests itself as accelerated growth of the heart. In addition, all of the above risk factors can lead to neuroendocrine failure and various changes in the valve located in the heart. It all starts with small deposits of fibrin crystals along the wall of the heart valve. After which thickening of the valve occurs due to the proliferation of fibrin fibers. This in turn leads to the appearance of a small area of ​​fibrosis on the valves, but this area expands quite quickly. All this sooner or later leads to the inability of the valve to perform its function. Fibrous changes usually begin from the aortic valve and gradually spread to the lungs. As mentioned earlier, in a third of all patients this disease will have a focal form. Often, manifestations of endocardial fibrosis become classic